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1.
Med. oral patol. oral cir. bucal (Internet) ; 28(1): e9-e15, ene. 2023. ilus, tab, graf
Artigo em Inglês | IBECS | ID: ibc-214878

RESUMO

experience, and oral hygiene status, and to measure salivary flow rate, salivary buffer capacity, and salivary cariogenic microflora with Marsh types. Material and Methods: A single-blind, prospective clinical study with 62 pediatric patients diagnosed with celiac disease with 64 controls. Clinical identification of molar-incisor hypomineralization (MIH) was followed according to the European Academy of Pediatric Dentistry criteria. DMFS and dfs index were used for the caries experience of each child. The clinical diagnosis of RAU was present or not. Oral hygiene was surveyed by recording the OHI-S and the CRT® Bacteria and Buffer Test was used to examine the cariogenic microflora of each child. Results: The prevalence of MIH was 61% and the number of recurrent aphthous ulcers were significantly higher in children with celiac disease. There was no statistically significant difference in the CD group, when DMFS, dfs, and MIH parameters were investigated according to dietary compliance. Higher dietary compliance resulted in better oral hygiene status. There was an inverse relationship between the duration of celiac diagnosis and the presence of MIH. A positive relation was found between the duration of the disease and the severity of MIH. In addition to the higher S. mutans counts, the salivary flow rate was very low in children with celiac disease, indicating a positive correlation between poor dietary compliance and poorer oral hygiene. Conclusions: In children, enamel defects and recurrent mucosal lesions may be a sign of celiac disease. Higher numbers of dental caries in permanent teeth of children with celiac disease may be related to Marsh 2 type. The pediatricians and/or pediatric gastroenterologists should refer the chin with celiac disease to the pediatric dentist for the accurate treatment of intraoral manifestations of the disease itself. (AU)


Assuntos
Humanos , Masculino , Feminino , Criança , Adolescente , Estomatite Aftosa , Cárie Dentária/epidemiologia , Doença Celíaca/complicações , Hipoplasia do Esmalte Dentário/epidemiologia , Estudos Prospectivos , Estudos de Casos e Controles
2.
Rev. esp. enferm. dig ; 114(11): 654-659, noviembre 2022. graf, tab
Artigo em Inglês | IBECS | ID: ibc-212284

RESUMO

Background and aim: to investigate the correlation betweenceliac disease and specific oral lesions.Methods: one hundred and fourteen pediatric patients wereenrolled and divided into three groups: celiac patients (CDgroup), patients with malabsorption without celiac disease(non-CD group) and healthy controls. Oral lesions of hardand soft tissues were detected and analyzed by Fisher’s testand odds ratio calculations.Results: a non-random correlation between the threegroups and the rate of enamel defects was detected. TheCD group showed more severe enamel defects compared tothe non-CD group. A non-random association between thethree groups and the number of mucous lesions was notobserved. Furthermore, malabsorption patterns not relatedto the celiac disease involved a relatively modest risk ofspecific enamel defects compared to controls.Conclusions: hypoplastic enamel defects of the permanentteeth could be reliable indicators for celiac disease. The etiology of specific hypoplastic enamel lesions mainly has tobe sought in the autoimmune response triggered by gluten, while the malabsorption would play only a minor role.Lesions of the oral mucous membranes should not be considered as specific risk indicators, however, they representa non-specific marker of malabsorption. (AU)


Assuntos
Humanos , Doença Celíaca/complicações , Hipoplasia do Esmalte Dentário , Glutens , Estudos Retrospectivos
3.
Av. odontoestomatol ; 37(2): 87-93, abr.-jun. 2021. ilus, tab
Artigo em Espanhol | IBECS | ID: ibc-217501

RESUMO

Introducción: El manejo clínico de las lesiones dentales ocasionadas por fluorosis dental se realiza mediante diversas técnicas que resultan ser muy agresivas y con pobres resultados estéticos, por estas razones se plantea como objetivo determinar el tratamiento mínimamente invasivo y estético para fluorosis dental en los estadios de 1 a 5 según el índice de Thylstrup and Fejerskov, descritos en la literatura. Metodología: Revisión sistemática de 2.299 artículos, procedentes de cuatro bases de datos: PubMed, Embase, Science Direct y EBSCO; la búsqueda se realizó con ocho términos MeSH y tres conectores booleanos para una selección final de 22 artículos en inglés, español y portugués, publicados entre enero de 2009 y diciembre de 2018. Resultados: El tratamiento más efectivo para lesiones en estadios TF1 y TF2 fue aclaramiento dental con peróxido de carbamida a 15% o peróxido de hidrógeno a 35% durante tres sesiones de 15 minutos cada una, reforzado con peróxido de carbamida a 10%. Para estadios TF3 y TF4 fue microabrasión con ácido clorhídrico al 6% y carburo de silicio y/o con aclaramiento dental. Para lesioneTF5 fue la técnica combinada de macro y microabrasión con ácido fosfórico a 37% o ácido clorhídrico a 6%, 15% y 18%, aclaramiento y aplicación de resina infiltrante. Conclusión: Existen bases científicas que indican que el tratamiento de la Fluorosis es directamente proporcional al estadio de la lesión. (AU)


Introduction: The clinical management of dental lesions caused by dental fluorosis is carried out through various techniques that are very aggressive and with poor aesthetic results. For these reasons, the aim is to determine the minimally invasive and aesthetic treatment for dental fluorosis in stages of 1 to 5 according to the Thylstrup and Fejerskov index, described in the literature. Methodology: Systematic review of 2,299 articles, from four databases: PubMed, Embase, Science Direct and EBSCO; the search was carried out with eight MeSHterms and three Boolean connectors for a final selection of 22 articles in English, Spanish and Portuguese, published between January 2009 and December2018. Results: The most effective treatment for lesions in stages TF1 and TF2 was dental clearance with carbamide peroxide at 15% or hydrogen peroxide at 35% for three sessions of 15 minutes each, reinforced with 10% carbamide peroxide. For stages TF3 and TF4 it was microabrasion with 6% hydrochloric acid and silicon carbide and / or with dental clearance. For lesioneTF5 was the combined technique of macro and microabrasion with phosphoric acid at 37% or hydrochloric acid at 6%, 15% and 18%, clearance and application of infiltrating resin. Conclusion: There are scientific bases that indicate that the treatment of Fluorosis is directly proportional to the stage of the lesion. (AU)


Assuntos
Humanos , Fluorose Dentária/tratamento farmacológico , Estética Dentária , Clareamento Dental , Hipoplasia do Esmalte Dentário
4.
Cient. dent. (Ed. impr.) ; 18(2): 111-118, abr. 2021. ilus, tab
Artigo em Espanhol | IBECS | ID: ibc-216977

RESUMO

El objetivo de esta revisión sistemática fue recoger los estudios que evalúan la relación entre problemas respiratorios en la infancia y el riesgo de desarrollar alteraciones del esmalte. Se realizó una búsqueda sistemática de la literatura publicada hasta el 1 de febrero de 2021 en las siguientes bases de datos: Pubmed, Scoupus, Web of Science y Cochrane. De un total de 173 artículos, 20 artículos cumplían los criterios de inclusión. Un total de 16 artículos estudiaron la relación con alteraciones respiratorias y otros 4 artículos la relación con fármacos frecuentemente administrados en estas patologías. Los estudios analizados muestran que los pacientes que han padecido asma u otras enfermedades respiratorias en los primeros años de vida tienen mayor riesgo de desarrollar alteraciones del esmalte, síndrome incisivo molar y/o segundo molar temporal hipomineralizado. Además, los fármacos empleados para su tratamiento también pueden ser un posible factor etiopatogénico de estas patologías. Debido a la limitada evidencia científica disponible, es necesaria la realización de nuevos estudios clínicos aleatorizados para confirmar la hipótesis planteada. (AU)


The aim of this systematic review was to collect studies that evaluate the relationship between respiratory problems in childhood and the risk of developing enamel disorders. A systematic search of the literature published up to 1 February 2021 was conducted in the following databases: Pubmed, Scoupus, Web of Science and Cochrane. Out of a total of 173 articles, 20 articles met the inclusion criteria. A total of 16 articles studied the relationship with respiratory disorders and a further 4 articles studied the relationship with drugs frequently administered in these pathologies. The studies analysed show that patients who have suffered from asthma or other respiratory diseases in their early years of life have a higher risk of developing enamel disorders, hypomineralised incisor-molar and/or second primary molar syndrome. In addition, the drugs used for their treatment may also be a possible etiopathogenic factor in these pathologies. Due to the limited scientific evidence available, new randomised clinical studies are needed to confirm the proposed hypothesis. (AU)


Assuntos
Humanos , Masculino , Feminino , Recém-Nascido , Lactente , Pré-Escolar , Criança , Hipoplasia do Esmalte Dentário , Esmalte Dentário , Doenças Respiratórias , Asma , Desmineralização do Dente
5.
Med. oral patol. oral cir. bucal (Internet) ; 23(6): e639-e645, nov. 2018. ilus, tab
Artigo em Inglês | IBECS | ID: ibc-176385

RESUMO

BACKGROUND: The objective of this study was to evaluate the dental and oral manifestations in patients with celiac disease. MATERIAL AND METHODS: The sample consisted of 40 patients with the disease and 40 without the disease matched by age in southern Brazil. The CD group included patients previously diagnosed by positive anti-endomysial (IgA) examination and confirmed by small intestine biopsy. The presence of dental enamel defects and dental caries was evaluated by a calibrated researcher according to AINE's and WHO's criteria, respectively. The history of recurrent aphthous ulcers and dry mouth was obtained through reporting. For the evaluation of the salivary flow, the saliva samples were obtained through the non-stimulated and stimulated saliva collection method. RESULTS: There was a significant association between CD and dental enamel defects (OR =2.38, P = 0.045) and dry mouth (OR = 9.15, P =0.002). No difference was found for the report of recurrent aphthous ulcers and caries experience between the two groups. Patients with CD had normal pattern of unstimulated and stimulated saliva flow rates (0.67 ± 0.38 ml / min and 1.14 ± 0.47 ml / min, respectively). A higher occurrence of dental enamel defects was observed in patients with classic CD (P = 0.054). Of the 1,962 permanent teeth, 59 presented dental enamel defects, 71.8% of which were in patients with CD (P = 0.001), predominantly in molars (P = 0.009). CONCLUSIONS: CD increased the likelihood of dental enamel defects and dry mouth sensation. The oral examination can be an important auxiliary tool for the identification of cases of the disease


Assuntos
Humanos , Masculino , Feminino , Adolescente , Doença Celíaca/complicações , Doenças da Boca/etiologia , Hipoplasia do Esmalte Dentário/etiologia , Fatores de Risco , Estudos de Casos e Controles
6.
Med. oral patol. oral cir. bucal (Internet) ; 21(5): e587-e594, sept. 2016. tab, graf
Artigo em Inglês | IBECS | ID: ibc-155770

RESUMO

BACKGROUND: Rare diseases (RD) are those that present a lower prevalence than 5 cases per 10.000 population. The main objective of this review was to study the effect on oral health in rare diseases, while the secondary objective of the study is theme upgrade. MATERIAL AND METHODS: Comparative observational case-control studies were analysed and a systematic review was conducted in PubMed. Each rare disease listed on the statistical data record of the Health Portal of the Ministry of Equality, Health and Social Policies Board of Andalusia was associated with 'oral health'. The variables studied included dental, oral mucosa and occlusion alterations, oral pathologies (caries, periodontal disease) and other alterations (mouth breathing, parafunctional habits, etc). A bias analysis of the variable caries was conducted. RESULTS: Six RD were selected through our inclusion and exclusion criteria (hypogammaglobulinemia, Rett syndrome, Marfan syndrome, Prader-Willi syndrome, cystic fibrosis and Cri du chat syndrome) in a total of 8 publications, of which four trials were classified as high risk of bias and one of them as medium risk. There were not trials with low risk of bias. CONCLUSIONS: The main statistically significant differences found by Syndrome compared to a control group were in Hypogammaglobulinemia with a greater tendency to enamel hypoplasia and dry mouth. The Rett syndrome had, as well, a greater tendency to an anterior open bite, ogival palate, bruxism, mouth breathing and tongue thrusting. Prader-Willi syndrome had a tendency of dental erosion, and Cri du chat syndrome showed a higher association to Tannerella forsythia


Assuntos
Humanos , Doenças Raras/complicações , Doenças da Boca/epidemiologia , Agamaglobulinemia/complicações , Hipoplasia do Esmalte Dentário/epidemiologia , Síndrome de Cri-du-Chat/complicações
7.
Cient. dent. (Ed. impr.) ; 12(3): 193-197, sept.-dic. 2015. ilus
Artigo em Espanhol | IBECS | ID: ibc-147161

RESUMO

El síndrome incisivo molar o MIh se describe como una hipomineralización de origen sistémico que afecta de 1 a 4 de los primeros molares permanentes y que se asocia frecuentemente con alteraciones de los incisivos, siendo su aparición cada vez más frecuente. Los dientes afectados van a presentar un aumento de su contenido proteico y una disminución del contenido mineral. El tratamiento del MIh supone un reto para el odontólogo. Las características clínicas de este desorden, como la ruptura posteruptiva o la formación de cavidades atípicas, complican la obtención de un resultado duradero. Los últimos estudios apuntan a que los sistemas adhesivos de autograbado consiguen una fuerza de adhesión mayor que otros sistemas. El pretratamiento de la lesión puede producir un aumento de la fuerza adhesiva, aunque los resultados observados no permiten estandarizar este procedimiento. Las opciones más prometedoras son el hipoclorito sódico y las resinas infiltrantes. La mejor opción terapéutica para el MIh actualmente consiste en la realización de una reconstrucción con resina compuesta, utilizando un sistema adhesivo de autograbado, valorando la posibilidad de realizar un pretratamiento con hipoclorito sódico o resinas infiltrantes (AU)


The molar incisor syndrome (MIh) is described as a hypomineralization of systemic origin affecting 1 to 4 of the first permanent molars that is frequently associated with alterations of the incisors, and its appearance is increasing. Affected teeth will file a raise of its protein content and decreased mineral content. The treatment of MIh is a challenge for the dentist. The clinical features of this disorder, as posteruptive rupture or the formation of atypical cavities complicate obtaining a lasting result. Recent studies suggest that selfetching adhesive systems achieve a greater adhesion force than other systems. Pretreatment of injury may result in increased adhesive strength, although the observed results do not allow to standardize this procedure. The most promising options are sodium hypochlorite and infiltrating resins. The best therapeutic option for MIh is currently performing a reconstruction with composite resin using a self-etching adhesive system, seeing the possibility of pretreatment with sodium hypochlorite or infiltrating resins (AU)


Assuntos
Humanos , Desmineralização do Dente/fisiopatologia , Dente Molar/fisiopatologia , Hipoplasia do Esmalte Dentário/fisiopatologia , Corrosão Dentária/métodos , Cimentos Dentários/uso terapêutico
8.
Cient. dent. (Ed. impr.) ; 12(3): 241-246, sept.-dic. 2015. ilus, tab
Artigo em Espanhol | IBECS | ID: ibc-147168

RESUMO

El propósito de este trabajo es conocer la evolución de la prevalencia del Síndrome Incisivo Molar, no sólo a nivel mundial sino también en nuestro ámbito nacional. Constituye una revisión bibliográfica sobre los artículos científicos publicados en relación con dicha alteración. Tras el análisis y teniendo en cuenta el año de publicación independientemente de la zona geográfica de estudio, no se observa un aumento de la prevalencia de hIM. Los países que experimentan mayores porcentajes son Australia, Brasil y Reino unido (44%, 40,2% y 40% respectivamente). Los que presentan menor prevalencia son China y Libia (2,8% y 2,9% respectivamente). En el ámbito nacional claramente se ha observado un incremento con el tiempo en la prevalencia del hIM. Las variaciones en los resultados obtenidos pueden reflejar diferencias reales entre regiones y países. Sin embargo, podrían explicarse, al menos en una parte, por las diferentes edades de la muestra, diferentes fechas de nacimiento, criterios de diagnóstico, variabilidad en cuanto a la posible etiología, entre otros factores (AU)


The purpose of this paper is to know the evolution of the prevalence of molar-incisor hypomineralization, both on a national and international scale, comprising a bibliographical review of the scientific papers published on the subject. Following the analysis and taking into consideration the year of publication, but not the geographical area of the study, an increase in the prevalence of MIh is not observed. The countries with the highest prevalence percentages are Australia, Brazil and the united Kingdom (with 44%, 40.2% and 40%, respectively). On the other hand, the countries with the lowest prevalence percentages are China and Libya, with 2.8% and 2.9%, respectively. On a national scale, a clear increment of the prevalence of MIh has been observed over time. Disparities in the obtained results show real differences between countries and regions. On one hand, they can be explained due to the different ages within the sample, dates of birth, diagnosis criteria and possible etiologies, among other factors (AU)


Assuntos
Humanos , Desmineralização do Dente/epidemiologia , Hipoplasia do Esmalte Dentário/epidemiologia , Doenças Dentárias/epidemiologia , Cárie Dentária/epidemiologia , Estudos Transversais
9.
Med. oral patol. oral cir. bucal (Internet) ; 18(2): 187-193, mar. 2013. ilus, tab
Artigo em Inglês | IBECS | ID: ibc-112384

RESUMO

Objective: The purpose of this study was to determine and compare the prevalence and presentations of developmental defects of the enamel (DDE) in the primary and permanent dentitions of a group of healthy schoolchildren residing in Granada (Spain).Study Design: A total of 1,414 healthy schoolchildren were examined using modified DDE criteria for recording enamel defects. Results: The prevalence of DDE of any type was 40.2% in primary dentition and 52% in permanent dentition (p<0.033). Of the 31,820 primary and permanent teeth examined in the study, 699 (4.1%) primary and 1,232 (8.3%) permanent teeth had some form of DDE. Diffuse opacity was the most common type of DDE observed in primary teeth, and demarcated opacity in the permanent teeth. Enamel hypoplasia was the least prevalent defect in both dentition types. Conclusions: The study population showed a high prevalence of DDE in primary as well as in permanent dentition, reflecting the current increasing trend of this condition, which should be considered as a significant public health problem (AU)


Assuntos
Humanos , Masculino , Feminino , Criança , Esmalte Dentário/anormalidades , Anormalidades Dentárias/epidemiologia , Hipoplasia do Esmalte Dentário/epidemiologia , Dente Decíduo/anormalidades , Estudos Transversais
10.
Av. odontoestomatol ; 28(6): 287-301, nov.-dic. 2012. ilus, tab
Artigo em Espanhol | IBECS | ID: ibc-110212

RESUMO

Las alteraciones del desarrollo embriológico de la dentición provocan anomalías y displasias dentarias. Los factores etiopatogénicos implicados en las alteraciones del desarrollo dentario son básicamente dos: genéticos y ambientales. Según la fase del desarrollo en que afecten al órgano del esmalte y a los tejidos dentarios, aparecerán diferentes anomalías y/o displasias dentales. El control genético del desarrollo dentario se lleva a cabo mediante dos procesos: a) control de la histogénesis del esmalte y la dentina, y b) la especificación del tipo, tamaño y posición de cada diente. La mutación de los genes implicados en la amelogénesis (AMELX, ENAM, MMP20 y KLK4) o en la dentinogénesis (DSPP) produce alteraciones del desarrollo dentario aisladas o no sindrómicas. Por el contrario, las mutaciones de los genes reguladores morfogenéticos involucrados en la determinación de la posición y el desarrollo precoz de los órganos dentarios (genes homeobox), además de alterar la morfodiferenciación dentaria, tienen efectos pleiotrópicos y afectan a otros órganos, provocando síndromes hereditarios en los que uno de sus rasgos es la alteración dentaria. En este artículo se revisan las principales anomalías y displasias dentarias de causa genético-hereditaria (AU)


Alterations of the embryologic development of the dentition cause dental anomalies and dysplasias. The causing factors involved in the disturbances of tooth development are basically two: genetic and environmental factors. Depending on the phase of tooth development when the factors act, they will appear different dental anomalies and dysplasias. Genetic control of tooth development is carried out through two processes: a) control of amelogenesis and dentinogenesis, and b) the specification of the type, size and position of each tooth. Mutation of genes involved in amelogenesis (AMELX, ENAM, MMP20 and KLK4) and dentinogenesis (DSPP) produces non-syndromic alterations of tooth development. On the contrary, mutations in morphogenetic regulatory genes involved in determining the position and the early development of the teeth (homeobox genes), not only alter teeth morpho differentiation, but also have pleiotropic effects affecting other organs, causing hereditary syndromes in which one of its features is the presence of dental abnormalities. Dental anomalies and dysplasias of genetic origin are reviewed in this article (AU)


Assuntos
Humanos , Anormalidades Dentárias/genética , Odontodisplasia/genética , Amelogênese/genética , Anodontia/genética , Dentinogênese , Doenças Raras/etiologia , Mutação/genética , Hipoplasia do Esmalte Dentário/genética
11.
Cient. dent. (Ed. impr.) ; 9(1): 49-53, ene.-abr. 2012. ilus
Artigo em Espanhol | IBECS | ID: ibc-104945

RESUMO

Introducción: La insuficiencia renal es un proceso que denota una pérdida de capacidad funcional de las nefronas. Se clasifica en aguda, subaguda y crónica basada en su forma de aparición y, sobretodo, en relación a las posibilidades de recuperación de la lesión estructural. A pesar de que la insuficiencia renal aguda es reversible en la mayoría de los casos, presenta un curso progresivo hacia la insuficiencia renal terminal, incluso aunque la causa de la nefropatía inicial desaparezca. La diálisis conduce a alteraciones sistémicas y complicaciones orales, y si a esto le sumamos la falta de cumplimiento de los hábitos de higiene oral por parte de muchos de estos pacientes, esta población presenta una prevalencia mayor de lesiones orales. Objetivo: El objetivo de esta revisión ha sido evaluar las alteraciones bucales de pacientes con insuficiencia renal. Métodos: Se realizó una búsqueda sistemática en Enero de 2012 en la base de datos PubMed con las palabras clave: renal failure, dentist, haemodialysis y oral health. Resultados: Los pacientes con insuficiencia renal crónica presentan mayor prevalencia de xerostomía, hiperplasiagingival, placa supragingival, gingivitis y mayor número de dientes con pérdida de inserción. Los niños con insuficiencia renal presentan un retraso en la erupción dental y alteraciones en el esmalte. Sin embargo, estos pacientes presentan una baja prevalencia de caries (AU)


Introduction: Renal insufficiency is a process that presents a loss of functional capacity of the nephrons. It is classified as acute, subacute and chronic based on the form of its appearance and, above all, in relation to the possibilities of recovery of the structural lesion. Despite the fact that acute renal insufficiency is reversible in the majority of the cases, it presents a progressive course towards terminal renal insufficiency, even though the cause of the initial nephropathy may disappear. Dialysis leads to systemic alterations and oral complications, and if we add to this the lack of compliance with good oral hygiene habits by many of these patients, this population presents a greater prevalence of oral lesions. Objective: The objective of this review has been to evaluate the oral alterations of patients with renal insufficiency. Methods: A systematic search was conducted in January 2012 of the PubMed database with the key words: renal failure, dentist, haemodialysis and oral health. Results: The patients with chronic renal insufficiency present greater prevalence of xerostomy, gingival hyperplasia, supragingival plaque, gingivitis and greater number of teeth with loss of attachment. Children with renal insufficiency present a delay in dental eruption and alterations in the enamel. However, these patients present a low prevalence of cavities (AU)


Assuntos
Humanos , Masculino , Feminino , Criança , Adulto , Insuficiência Renal Crônica/complicações , Doenças da Boca/etiologia , Diálise Renal/efeitos adversos , Periodontite/diagnóstico , Hiperplasia Gengival/diagnóstico , Hipoplasia do Esmalte Dentário/diagnóstico
12.
Med. oral patol. oral cir. bucal (Internet) ; 16(4): 478-481, jul. 2011. tab, ilus
Artigo em Inglês | IBECS | ID: ibc-93035

RESUMO

Introduction: Tuberous sclerosis complex (TSC) is a neurocutaneous syndrome produced by a number of geneticmutations. The disease is characterized by the development of benign tumors affecting different body systems.The most common oral manifestations of TSC are fibromas, gingival hyperplasia and enamel hypoplasia.Clinical Case: A 35-year-old woman diagnosed with TSC presented with a reactive fibroma of considerable sizeand rapid growth in the region of the right lower third molar.Discussion: In the present case the association of TSC with dental malpositioning gave rise to a rapidly evolvingreactive fibroma of considerable diameter. Few similar cases can be found in the literature. Patients with TSCpresent mutations of the TSC1 and TSC2 genes, which intervene in cell cycle regulation and are important foravoiding neoplastic processes. No studies have been found associating TSC with an increased risk of oral cancer,though it has been shown that the over-expression of TSC2 could exert an antitumor effect. Careful oral and dentalhygiene, together with regular visits to the dentist, are needed for the prevention and early detection of any typeof oral lesion. The renal, pulmonary and cardiac alterations often seen in TSC must be taken into account for thecorrect management of these patients (AU)


Assuntos
Humanos , Feminino , Adulto , Esclerose Tuberosa/complicações , Hipoplasia do Esmalte Dentário/etiologia , Fibroma/etiologia , Hiperplasia Gengival/etiologia , Higiene Bucal
13.
Cient. dent. (Ed. impr.) ; 5(3): 239-246, sept.-dic. 2008. ilus, tab
Artigo em Es | IBECS | ID: ibc-70812

RESUMO

Con la denominación de Amelogénesis Imperfecta(AI) se define un grupo de enfermedades hereditarias heterogéneas clínica y genéticamente que se caracterizan por alteraciones del esmalte. También pueden observarse otras alteraciones orales y extraorales. La maloclusión más frecuente en estos pacientes es la mordida abierta. Algunos casos forman parte de un síndrome. Hasta el momento se han identificado mutaciones en cinco genes, AMELX, ENAM, KLK4, MMP20 yDLX3, que participan en la formación del esmalte normal, pero quedan otros por identificar. Los autores actualizan los conocimientos sobre la etiopatogenia, clasificación, manifestaciones clínicas, diagnóstico y tratamiento interdisciplinar de la AI (AU)


Amelogenesis imperfecta (AI) is a collective designation for a clinically and genetically diverse group of disorders displaying enamel malformations. Other oral and extraoral aberrations have been reported, and open bite is the most common malocclusion. Some cases present as a part of a syndrome. Mutations in five genes involved in normal enamel formation(AMELX, ENAM, MMP20, KLK4 and DLX3) have been identified as cause of amelogenesis imperfecta, but some others remain to be identified. This paper reviews current knowledge about etiopathogenesis, classification, clinical manifestations, diagnosis and interdisciplinary treatment of AI (AU)


Assuntos
Humanos , Masculino , Feminino , Amelogênese Imperfeita/diagnóstico , Amelogênese Imperfeita/epidemiologia , Amelogênese Imperfeita/etiologia , Hipoplasia do Esmalte Dentário/diagnóstico , Hipoplasia do Esmalte Dentário/epidemiologia , Hipoplasia do Esmalte Dentário/fisiopatologia , Diagnóstico Diferencial , Amelogênese Imperfeita/complicações , Amelogênese Imperfeita/fisiopatologia , Má Oclusão/complicações
15.
Cient. dent. (Ed. impr.) ; 4(1): 23-27, ene.-abr. 2007. ilus
Artigo em Es | IBECS | ID: ibc-053554

RESUMO

Introducción: La presente investigación ha sido realizada para conocer las manchas blancas del esmalte, su epidemiología, clasificación, forma de presentación y posible etiología, dentro del conjunto de discoloraciones dentarias. Objetivos: 1) Revisar las alteraciones del desarrollo del esmalte que pueden cursar con la aparición de manchas blancas en su superficie. 2) Determinar la prevalencia de manchas blancas. 3) Delimitar las localizaciones más frecuentes. 4) Identificar los posibles agentes causales de las mismas. Material y método: Para ello, se ha realizado un estudio descriptivo utilizando una muestra de 246 individuos (127 niñas y 119 niños) con edades comprendidas entre los tres y catorce años. Resultados: Se encontró que en el 23% de la muestra aparecía al menos una alteración de este tipo y con más frecuencia en niñas. El 93% se correspondía con opacidades, siendo el canino el diente más afectado en dentición temporal, mientras que en la permanente fue el incisivo central superior. Conclusiones: Los resultados obtenidos se compararon con los obtenidos por otros autores, llegando a las siguientes conclusiones: 1) El sexo no parece ser un factor determinante claro en la aparición de estas alteraciones; 2) Los dientes más afectados son los primeros molares e incisivos centrales superiores permanentes y los caninos temporales, tanto en su tercio medio como en el incisal; 3) Serán necesarios más estudios para establecer la prevalencia y la posible etiología (AU)


Introduction: The present investigation has been made to know spots enamel, their epidemiology, classification, form of presentation and possible etiology, within the set of dental discoloration. Objetives: 1) Revise the alterations of the development of the enamel that can study with the appearance of white stains in their superficial. 2) Determine the prevalence of white spots. 3) Define the more localizations frequence. 4) Identify the casual possible agents of the same ones. Material y method: For it has been made a descriptive study using a sample of 246 children (127 girls and 119 boys) between three and fourteen years old. Results: At 93% of the sample appear at least one alteration of this type and more frequently in girls. The 93% were opacities, being canine the most affected in temporal teeth, whereas in the permanent one it was the superior central incisor. Conclusions: The obtained results were compared with the obtains ones by others authors, reaching the following conclusions: 1) Sex does not seen to be clear a determinig factor in the appareance of alterations; 2) The teeth more affected are first molars and superior incisive permanent and temporal canine; 3) More studies will be necessary to establish the prevalence and possible etiology (AU)


Assuntos
Masculino , Feminino , Pré-Escolar , Criança , Adolescente , Humanos , Anormalidades Dentárias/epidemiologia , Esmalte Dentário/fisiopatologia , Hipoplasia do Esmalte Dentário/diagnóstico , Fluorose Dentária/diagnóstico , Desmineralização do Dente/diagnóstico
16.
Med. oral patol. oral cir. bucal (Internet) ; 11(1): 40-43, ene. 2006. ilus
Artigo em Es | IBECS | ID: ibc-042626

RESUMO

Objetivos: El objetivo del presente trabajo consistió en definir los patrones morfológicos y de calcificación en el esmalte de dientes afectados con amelogénesis imperfecta hipomineralizada, tras la aplicación del grabado ácido, mediante la utilización de la microscopía electrónica analítica cuantitativa.Diseño del estudio: Se estudian 5 fragmentos de caninos humanos permanentes de pacientes con amelogénesis imperfecta hipomineralizada y 5 caninos controles. Todas las muestras fueron tratadas con ácido fosfórico y posteriormente procesadas para su estudio morfológico y microanalítico. Resultados: Se observan dos tipos de patrones de grabados, I y II, así como áreas o islotes de patrón I insertados en extensas áreas homogéneas en las que predomina el patrón II. No se aprecian diferencias significativas en la concentración de calcio entre las muestras con amelogénesis imperfecta hipomineralizada y control después del grabado ácido tras el estudio microanalíticocuantitativo. El patrón tipo III no fue observado. Conclusiones: Se describen los cambios y distribución en la estructura del esmalte después del grabado ácido durante 30 segundosen dientes con amelogénesis imperfecta hipomineralizada. Aunque los datos obtenidos coinciden con alteraciones en el desarrollo de los prismas, no se detectan alteraciones en la concentración de calcio


Objectives: The purpose of this study was to use quantitative x-ray microprobe analysis with scanning electron microscopy to define the morphostructural and calcification patterns in the enamel of teeth with the hypomineralized variant of amelogenesis imperfecta. Study design: We compared 5 fragments of permanent human canines from patients with clinically diagnosed hypomineralized amelogenesis imperfecta and 5 normal permanent canines from subjects without amelogenesis imperfecta. All specimens were etched with phosphoric acid for morphological and microanalytical examination. Results: Two types of etching patterns were found; in addition, islets of pattern I were seen within areas of pattern II. Microanalysisdetected no significant differences in calcium concentration between specimens with amelogenesis imperfecta and normal control specimens after acid etching. Pattern III was not observed. Conclusions: The changes and their distribution in the enamel structure after 30 s of acid etching are described in teeth with this rare disorder. Although these data seem to coincide with alterations in prism development, no alterations in calcium concentrationwere found


Assuntos
Humanos , Hipoplasia do Esmalte Dentário/patologia , Condicionamento Ácido do Dente , Esmalte Dentário , Cálcio/análise , Estudos de Casos e Controles , Cristalização , Dente Canino , Hipoplasia do Esmalte Dentário/ultraestrutura , Microanálise por Sonda Eletrônica , Microscopia Eletrônica de Varredura , Esmalte Dentário/patologia , Esmalte Dentário/ultraestrutura , Ácidos Fosfóricos/farmacologia
17.
Pediátrika (Madr.) ; 25(1): 1-5, ene. 2005. ilus
Artigo em Es | IBECS | ID: ibc-036816

RESUMO

El esmalte dentario es uno de los tejidos más duros del organismo, sin embargo las células formadoras del esmalte, los ameloblastos, son muy sensibles a los cambios que ocurren durante el periodo de formación del diente. Son muchas las patologías que pueden alterar el funcionamiento del ameloblasto provocando lesiones de hipoplasia o hipomineralización, y a diferencia de lo que ocurre en otros tejidos, el esmalte dentario carece de mecanismos de reparación. En este artículo, revisamos las principales causas genéticas y ambientales que pueden ser responsables de las anomalías en esmalte dentario, así como el aspecto clínico que pueden presentar estas anomalías


Dental enamel is one of the hardest tissues in thehuman body. However, ameloblast cels wich compraisthe enamel, are extremely sensitive to the changes occurring during dental development. There are many pathologys that can alter the normal function in ameloblast cels and cause hypoplasia orhypomineralization. In contrast to other tissue enamel tissue does not have its own repair mechanisms. In this article we review the principal genetic andenvirolmental causes that may contribute to dental anomalies as well as the clinical aspect of these anomalies


Assuntos
Criança , Humanos , Esmalte Dentário/anormalidades , Hipoplasia do Esmalte Dentário/fisiopatologia , Amelogênese Imperfeita/fisiopatologia , Fluorose Dentária/fisiopatologia
18.
Rev. esp. cir. oral maxilofac ; 26(4): 240-244, jul.-ago. 2004. ilus
Artigo em En | IBECS | ID: ibc-36919

RESUMO

Los traumatismos que afectan a los cóndilos mandibulares pueden ocasionar una anquilosis de la articulación temporomandibular que, si se produce en edad de crecimiento, puede dar lugar a una deformidad facial con asimetría o retrognatia. Entre los diferentes tratamientos utilizados, la distracción osteogénica ofrece unas ventajas, entre ellas la posibilidad de aplicación en la infancia y la elongación de tejidos blandos. Presentamos un caso de anquilosis temporomandibular bilateral en una niña de 9 años, que tratamos con distracción extraoral tras resección del bloque anquilótico, pero sólo con la finalidad de elongar los tejidos blandos y mantener el espacio en la nueva cavidad glenoidea para evitar la reanquilosis y favorecer la remodelación del cóndilo (AU)


Assuntos
Feminino , Pré-Escolar , Humanos , Anquilose/diagnóstico , Anquilose/terapia , Artroplastia/métodos , Côndilo Mandibular/patologia , Côndilo Mandibular , Articulação Temporomandibular/patologia , Articulação Temporomandibular , Osteogênese Imperfeita/diagnóstico , Osteogênese Imperfeita/terapia , Assimetria Facial/complicações , Assimetria Facial/diagnóstico , Retrognatismo/complicações , Retrognatismo/diagnóstico , Hipoplasia do Esmalte Dentário/complicações , Hipoplasia do Esmalte Dentário/diagnóstico , Hipoplasia do Esmalte Dentário/terapia
19.
Artigo em Es | IBECS | ID: ibc-37988

RESUMO

Introducción: los niños nacidos en condiciones de alto riesgo presentan mayor riesgo de padecer enfermedades. A nivel oral observamos en estudios previos hipoplasias de esmalte, caries y alteraciones en paladar, fundamentalmente. Nuestro objetivo fue conocer la patología orofacial y su relación con el bajo peso al nacimiento en una población de niños nacidos en condiciones de alto riesgo de Granada. Pacientes y método: la muestra fue de 60 niños de 46 años nacidos en condiciones de alto riesgo y que necesitaron cuidados intensivos neonatales. Se realizó un estudio descriptivo para determinar la patología en estos niños y un análisis estadístico univariante para analizar las variables según el peso en el nacimiento. Resultados: observamos una relación estadísticamente significativa entre el peso al nacimiento menor de 1550 gramos y las variables perinatales. El porcentaje de anomalías estructurales dentarias, caries, hábitos orofaciales y traumatismos observado en nuestra muestra fue elevado, existiendo relación estadísticamente significativa con el peso al nacer. Los hábitos higiénicos y asistencia dental previa fueron escasos, no estando relacionados con el peso en el nacimiento. Conclusiones: es importante considerar los datos neonatales ya que aportan información sobre la presencia de secuelas en el paciente infantil nacido en condiciones de riesgo y permitirá al odontopediatra establecer protocolos preventivos y de atención al niño (AU)


Assuntos
Feminino , Pré-Escolar , Masculino , Criança , Humanos , Doenças da Boca/epidemiologia , Recém-Nascido de Baixo Peso , Cárie Dentária/epidemiologia , Anormalidades Maxilomandibulares/epidemiologia , Hipoplasia do Esmalte Dentário/epidemiologia
20.
Ortod. esp. (Ed. impr.) ; 43(2): 85-93, abr. 2003. ilus, tab
Artigo em Es | IBECS | ID: ibc-25804

RESUMO

La amelogénesis imperfecta constituye un grupo de defectos hereditarios que afectan al esmalte. La clasificación más reciente diferencia hasta cuatro tipos basándose en criterios clínicos, radiográficos, genéticos e histológicos. Las características clínicas son variadas dependiendo del tipo. Desde el punto de vista ortodóncico la amelogénesis imperfecta presenta una serie de dificultades añadidas en el manejo de los pacientes afectados, como son presentar maloclusiones severas, que fundamentalmente han sido asociadas con mordidas abiertas. Además existen dificultades en la adhesión de tubos y brackets así como problemas derivados del control de la higiene. Se describe el caso de tres hermanos afectados por amelogénesis imperfecta. (AU)


Assuntos
Adolescente , Feminino , Masculino , Humanos , Amelogênese Imperfeita/diagnóstico , Hipoplasia do Esmalte Dentário/diagnóstico , Amelogênese Imperfeita/complicações , Má Oclusão/complicações
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