RESUMO
Tests of a slow-release molluscicide containing 50 percent copper sulfate were undertaken in laboratory and field situations in St. Lucia. In laboratory trials, a granule form of the molluscicide produced 100 percent mortality of Biomphalaria glabrata down to 4 mg.liter active ingredient (a.i), while the pellet form produced 100 percent mortality down to 8 mg/liter a.i. In field trials, a dose of 100 mg/liter a.i. in granule form caused mortality of B. glabrata in banana drains but had no effect on B. glabrata populations in a marsh habitat. In both habitats, the dose of 100mg/liter produced mortality of other molluscan fauna which caused changes in the molluscan diversity indices. This failure in field trials may have been due to dilution of copper levels caused by flooding and also by uptake of copper by mud and algae (AU)
Assuntos
21003 , Moluscocidas , Cobre , Eucariotos , Biomphalaria , População , Sulfatos , Fatores de Tempo , Santa LúciaAssuntos
Adolescente , Adulto , Idoso , Criança , Feminino , Humanos , Masculino , Pessoa de Meia-Idade , Úlcera/tratamento farmacológico , Úlcera Varicosa/tratamento farmacológico , Zinco/uso terapêutico , Administração Oral , Doença Crônica , Ensaios Clínicos como Assunto , Placebos , Sulfatos/uso terapêutico , Zinco/administração & dosagem , JamaicaRESUMO
The acid excretion of patients with sickle-cell anaemia has been studied. There is a mild defect in urinary acidification and a decreased H+ excretion in response to ammonium chloride loading. The acidification defect was not corrected by oral administration of a phosphate solution. Infusion of sodium sulphate solution in subjects who were avidly reabsorbing sodium produced equal degrees of urine acidification in patients and controls. Studies on bicarbonate reabsorption were inconclusive. We conclude that patients with sickle-cell anaemia have a mild form of incomplete distal renal tubular acidosis (AU)
