Tumor necrosis factor-like weak inducer of apoptosis and its potential roles in lupus nephritis.
Liu, Zhi-Chun; Zhou, Qiao-Ling.
; 61(4): 277-84, 2012 Apr.
Artigo em Inglês | MEDLINE | ID: mdl-22297307
TWEAK/Fn14 Activation Participates in Skin Inflammation.
TWEAK as a target for therapy in systemic lupus erythematosus.
TWEAK/Fn14 axis: the current paradigm of tissue injury-inducible function in the midst of complexities.
Targeting of Fn14 Prevents Cancer-Induced Cachexia and Prolongs Survival.
[Expression of TWEAK/Fn14 in Pancreatic Cancer].
Binding efficiency of protein-protein complexes.
Human platelets contain and release TWEAK.
TWEAK/Fn14 interaction induces proliferation and migration in human airway smooth muscle cells via activating the NF-κB pathway.
Fn14-Fc suppresses germinal center formation and pathogenic B cells in a lupus mouse model via inhibition of the TWEAK/Fn14 Pathway.
TWEAK/Fn14 activation induces keratinocyte proliferation under psoriatic inflammation.