Antagonistic regulation of apoptosis and differentiation by the Cut transcription factor represents a tumor-suppressing mechanism in Drosophila.
Zhai, Zongzhao; Ha, Nati; Papagiannouli, Fani; Hamacher-Brady, Anne; Brady, Nathan; Sorge, Sebastian; Bezdan, Daniela; Lohmann, Ingrid.
; 8(3): e1002582, 2012.
Artigo em Inglês | MEDLINE | ID: mdl-22438831
The regulation and evolution of a genetic switch controlling sexually dimorphic traits in Drosophila.
Low affinity binding site clusters confer hox specificity and regulatory robustness.
A system of repressor gradients spatially organizes the boundaries of Bicoid-dependent target genes.
The Hox proteins Ubx and AbdA collaborate with the transcription pausing factor M1BP to regulate gene transcription.
Control of nerve cord formation by Engrailed and Gooseberry-Neuro: A multi-step, coordinated process.
Distinct subsets of Eve-positive pericardial cells stabilise cardiac outflow and contribute to Hox gene-triggered heart morphogenesis in .
A feed-forward relay integrates the regulatory activities of Bicoid and Orthodenticle via sequential binding to suboptimal sites.
A Hox Transcription Factor Collective Binds a Highly Conserved Distal-less cis-Regulatory Module to Generate Robust Transcriptional Outcomes.
Impacts of the ubiquitous factor Zelda on Bicoid-dependent DNA binding and transcription in Drosophila.
Cooperative recruitment of Yan via a high-affinity ETS supersite organizes repression to confer specificity and robustness to cardiac cell fate specification.