Mutations in Interleukin-10 Receptor and Clinical Phenotypes in Patients with Very Early Onset Inflammatory Bowel Disease: A Chinese VEO-IBD Collaboration Group Survey.
Huang, Zhiheng; Peng, Kaiyue; Li, Xiaoqin; Zhao, Ruiqin; You, Jieyu; Cheng, Xiuyong; Wang, Zhaoxia; Wang, Ying; Wu, Bingbing; Wang, Huijun; Zeng, Huasong; Yu, Zhuowen; Zheng, Cuifang; Wang, Yuesheng; Huang, Ying.
Inflamm Bowel Dis
; 23(4): 578-590, 2017 Apr.
Artigo em Inglês | MEDLINE | ID: mdl-28267044
Novel exonic mutation inducing aberrant splicing in the IL10RA gene and resulting in infantile-onset inflammatory bowel disease: a case report.
IL-10 in humans: lessons from the gut, IL-10/IL-10 receptor deficiencies, and IL-10 polymorphisms.
Very early-onset inflammatory bowel disease (IBD) in infancy is a different disease entity from adult-onset IBD; one form of interleukin-10 receptor mutations.
Loss of interleukin-10 signaling and infantile inflammatory bowel disease: implications for diagnosis and therapy.
Clinical features of interleukin 10 receptor gene mutations in children with very early-onset inflammatory bowel disease.
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Inflammatory bowel disease and mutations affecting the interleukin-10 receptor.
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Identification of the Glycosaminoglycan Binding Site of Interleukin-10 by NMR Spectroscopy.
Evidence for non-neutralizing autoantibodies against IL-10 signalling components in patients with inflammatory bowel disease.