MicroRNA-200c inhibits epithelial-mesenchymal transition, invasion, and migration of lung cancer by targeting HMGB1.
Liu, Po-Len; Liu, Wei-Lun; Chang, Jia-Ming; Chen, Yung-Hsiang; Liu, Yu-Peng; Kuo, Hsuan-Fu; Hsieh, Chong-Chao; Ding, Yu-Sian; Chen, Wei-Wei; Chong, Inn-Wen.
; 12(7): e0180844, 2017.
Artigo em Inglês | MEDLINE | ID: mdl-28727734
Prediction of potential drivers connecting different dysfunctional levels in lung adenocarcinoma via a protein-protein interaction network.
[Role of miR-155 in invasion and metastasis of lung adenocarcinoma A549 cells].
miR-1236-3p suppresses the migration and invasion by targeting KLF8 in lung adenocarcinoma A549 cells.
Targeting the Wnt-Regulatory Protein CTNNBIP1 by microRNA-214 Enhances the Stemness and Self-Renewal of Cancer Stem-Like Cells in Lung Adenocarcinomas.
MicroRNA-29c functions as a tumor suppressor by targeting VEGFA in lung adenocarcinoma.
MiRNA-145 suppresses lung adenocarcinoma cell invasion and migration by targeting N-cadherin.
Conceptual and Experimental Tools to Understand Spatial Effects and Transport Phenomena in Nonlinear Biochemical Networks Illustrated with Patchy Switching.
The association between miR-34 dysregulation and distant metastases formation in lung adenocarcinoma.
The LncRNA NEAT1 Accelerates Lung Adenocarcinoma Deterioration and Binds to Mir-193a-3p as a Competitive Endogenous RNA.
Clinical Value and Prospective Pathway Signaling of MicroRNA-375 in Lung Adenocarcinoma: A Study Based on the Cancer Genome Atlas (TCGA), Gene Expression Omnibus (GEO) and Bioinformatics Analysis.