Magnolol protects against ischemic-reperfusion brain damage following oxygen-glucose deprivation and transient focal cerebral ischemia.
Huang, Sheng-Yang; Tai, Shih-Huang; Chang, Che-Chao; Tu, Yi-Fang; Chang, Chih-Han; Lee, E-Jian.
Int J Mol Med
; 41(4): 2252-2262, 2018 Apr.
Artigo em Inglês | MEDLINE | ID: mdl-29336466
Preconditioning with recombinant high-mobility group box 1 induces ischemic tolerance in a rat model of focal cerebral ischemia-reperfusion.
Neuroprotective hypothermia - Why keep your head cool during ischemia and reperfusion.
Upregulation of NOX2 and NOX4 Mediated by TGF-ß Signaling Pathway Exacerbates Cerebral Ischemia/Reperfusion Oxidative Stress Injury.
p53 opens the mitochondrial permeability transition pore to trigger necrosis.
IL-35 is a Protective Immunomodulator in Brain Ischemic Injury in Mice.
Assessment at the single-cell level identifies neuronal glutathione depletion as both a cause and effect of ischemia-reperfusion oxidative stress.
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Mitofusin 2 Exerts a Protective Role in Ischemia Reperfusion Injury Through Increasing Autophagy.
The Effect of 3',4'-Dihydroxyflavonol on Lipid Peroxidation in Rats with Cerebral Ischemia Reperfusion Injury.
All-Trans Retinoic Acid Ameliorates the Early Experimental Cerebral Ischemia-Reperfusion Injury in Rats by Inhibiting the Loss of the Blood-Brain Barrier via the JNK/P38MAPK Signaling Pathway.