Human iPSC-MSCs prevent steroid-resistant neutrophilic airway inflammation via modulating Th17 phenotypes.
Fang, Shu-Bin; Zhang, Hong-Yu; Jiang, Ai-Yun; Fan, Xing-Liang; Lin, Yong-Dong; Li, Cheng-Lin; Wang, Cong; Meng, Xiang-Ci; Fu, Qing-Ling.
Stem Cell Res Ther
; 9(1): 147, 2018 May 24.
Artigo em Inglês | MEDLINE | ID: mdl-29793557
Heme oxygenase-1 exerts a protective role in ovalbumin-induced neutrophilic airway inflammation by inhibiting Th17 cell-mediated immune response.
[B Cell Activating Transcription Factor Regulates Acute Airway Inflammation in Asthmatic Mice].
Transcription factors GATA-3 and RORÎ³t are important for determining the phenotype of allergic airway inflammation in a murine model of asthma.
CCL2/CCR2-dependent recruitment of Th17 cells but not Tc17 cells to the lung in a murine asthma model.
Lipopolysaccharides promote a shift from Th2-derived airway eosinophilic inflammation to Th17-derived neutrophilic inflammation in an ovalbumin-sensitized murine asthma model.
[Expression and role of Tc17 cells in mice with neutrophilic asthma].
[RORÎ³t expression in the pulmonary tissue of asthmatic mice and the inhibitory effects of budesonide].
Innate lymphoid cells in asthma: Will they take your breath away?
Molecular Mechanisms of Airway Hyperresponsiveness in a Murine Model of Steroid-Resistant Airway Inflammation.
Oral administration of Saccharomyces cerevisiae UFMG A-905 prevents allergic asthma in mice.