A recessive form of hyper-IgE syndrome by disruption of ZNF341-dependent STAT3 transcription and activity.

Béziat, Vivien; Li, Juan; Lin, Jian-Xin; Ma, Cindy S; Li, Peng; Bousfiha, Aziz; Pellier, Isabelle; Zoghi, Samaneh; Baris, Safa; Keles, Sevgi; Gray, Paul; Du, Ning; Wang, Yi; Zerbib, Yoann; Lévy, Romain; Leclercq, Thibaut; About, Frédégonde; Lim, Ai Ing; Rao, Geetha; Payne, Kathryn; Pelham, Simon J; Avery, Danielle T; Deenick, Elissa K; Pillay, Bethany; Chou, Janet; Guery, Romain; Belkadi, Aziz; Guérin, Antoine; Migaud, Mélanie; Rattina, Vimel; Ailal, Fatima; Benhsaien, Ibtihal; Bouaziz, Matthieu; Habib, Tanwir; Chaussabel, Damien; Marr, Nico; El-Benna, Jamel; Grimbacher, Bodo; Wargon, Orli; Bustamante, Jacinta; Boisson, Bertrand; Müller-Fleckenstein, Ingrid; Fleckenstein, Bernhard; Chandesris, Marie-Olivia; Titeux, Matthias; Fraitag, Sylvie; Alyanakian, Marie-Alexandra; Leruez-Ville, Marianne; Picard, Capucine; Meyts, Isabelle.
Sci Immunol; 3(24)2018 Jun 15.
Artigo em Inglês | MEDLINE | ID: mdl-29907691