Up-Regulation Thioredoxin Inhibits Advanced Glycation End Products-Induced Neurodegeneration.
Ren, Xiang; Wang, Ni-Na; Qi, Hui; Qiu, Yuan-Yuan; Zhang, Cheng-Hong; Brown, Emily; Kong, Hui; Kong, Li.
Cell Physiol Biochem
; 50(5): 1673-1686, 2018.
Artigo em Inglês | MEDLINE | ID: mdl-30384364
Epigenetic regulation of the thioredoxin-interacting protein (TXNIP) gene by hyperglycemia in kidney.
Cytokines Regulate ß-Cell Thioredoxin-interacting Protein (TXNIP) via Distinct Mechanisms and Pathways.
W2476 ameliorates ß-cell dysfunction and exerts therapeutic effects in mouse models of diabetes via modulation of the thioredoxin-interacting protein signaling pathway.
Deletion of thioredoxin-interacting protein improves cardiac inotropic reserve in the streptozotocin-induced diabetic heart.
[The renal protective effect and mechanism of liraglutide in diabetic mice induced by high-fat diet].
Thioredoxin-interacting protein mediates dysfunction of tubular autophagy in diabetic kidneys through inhibiting autophagic flux.
Variations of thioredoxin system contributes to increased susceptibility to apoptosis in cardiomyocytes of type 2 diabetic rats.
Thioredoxin-Interacting Protein (TXNIP) Suppresses Expression of Glutamine Synthetase by Inducing Oxidative Stress in Retinal Muller Glia Under Diabetic Conditions.
Skin changes in streptozotocin-induced diabetic rats.
Functional changes in the neural retina occur in the absence of mitochondrial dysfunction in a rodent model of diabetic retinopathy.