PFKFB3 promotes endotoxemia-induced myocardial dysfunction through inflammatory signaling and apoptotic induction.
Tian, Wen; Guo, Hong-Sheng; Li, Chong-Yao; Cao, Wei; Wang, Xue-Ying; Mo, Dan; Hao, Xiao-Wei; Feng, Ying-Da; Sun, Yang; Lei, Fan; Zhang, Hui-Nan; Zhao, Ming-Gao; Li, Xiao-Qiang.
Toxicol Appl Pharmacol
; 368: 26-36, 2019 Apr 01.
Artigo em Inglês | MEDLINE | ID: mdl-30776389
Exercise-Induced Changes in Glucose Metabolism Promote Physiological Cardiac Growth.
Small molecule inhibition of 6-phosphofructo-2-kinase suppresses t cell activation.
Expression of PFKFB3 and Ki67 in lung adenocarcinomas and targeting PFKFB3 as a therapeutic strategy.
Inhibition of 6-phosphofructo-2-kinase (PFKFB3) suppresses glucose metabolism and the growth of HER2+ breast cancer.
Role of PFKFB3-driven glycolysis in vessel sprouting.
Endothelial PFKFB3 plays a critical role in angiogenesis.
Acceleration of the glycolytic flux by steroid receptor coactivator-2 is essential for endometrial decidualization.
Type 2 Diabetes Dysregulates Glucose Metabolism in Cardiac Progenitor Cells.
PFKFB3 activation in cancer cells by the p38/MK2 pathway in response to stress stimuli.
The Glycolytic Enzyme PFKFB3 Is Involved in Estrogen-Mediated Angiogenesis via GPER1.