Distinct phenotype of CD4<sup>+</sup> T cells driving celiac disease identified in multiple autoimmune conditions.
Christophersen, Asbjørn; Lund, Eivind G; Snir, Omri; Solà, Elsa; Kanduri, Chakravarthi; Dahal-Koirala, Shiva; Zühlke, Stephanie; Molberg, Øyvind; Utz, Paul J; Rohani-Pichavant, Mina; Simard, Julia F; Dekker, Cornelia L; Lundin, Knut E A; Sollid, Ludvig M; Davis, Mark M.
; 25(5): 734-737, 2019 05.
Artigo em Inglês | MEDLINE | ID: mdl-30911136
Celiac disease: Autoimmunity in response to food antigen.
Integration of genetic and immunological insights into a model of celiac disease pathogenesis.
The composition and differentiation potential of the duodenal intraepithelial innate lymphocyte compartment is altered in coeliac disease.
Healthy HLA-DQ2.5+ Subjects Lack Regulatory and Memory T Cells Specific for Immunodominant Gluten Epitopes of Celiac Disease.
Classification and management of refractory coeliac disease.
Gluten-specific T cells cross-react between HLA-DQ8 and the HLA-DQ2α/DQ8ß transdimer.
Celiac disease: an immunological jigsaw.
Experimental hookworm infection and gluten microchallenge promote tolerance in celiac disease.
HLA-DQ2.5 genes associated with celiac disease risk are preferentially expressed with respect to non-predisposing HLA genes: Implication for anti-gluten T cell response.
HLA-DQ:gluten tetramer test in blood gives better detection of coeliac patients than biopsy after 14-day gluten challenge.