The immunoproteasome catalytic ß5i subunit regulates cardiac hypertrophy by targeting the autophagy protein ATG5 for degradation.
Xie, Xin; Bi, Hai-Lian; Lai, Song; Zhang, Yun-Long; Li, Nan; Cao, Hua-Jun; Han, Ling; Wang, Hong-Xia; Li, Hui-Hua.
; 5(5): eaau0495, 2019 May.
Artigo em Inglês | MEDLINE | ID: mdl-31086810
A proliferative burst during preadolescence establishes the final cardiomyocyte number.
Understanding Key Mechanisms of Exercise-Induced Cardiac Protection to Mitigate Disease: Current Knowledge and Emerging Concepts.
No Evidence for Cardiomyocyte Number Expansion in Preadolescent Mice.
Differential Role of G Protein-Coupled Receptor Kinase 5 in Physiological Versus Pathological Cardiac Hypertrophy.
miR-106a promotes cardiac hypertrophy by targeting mitofusin 2.
Long noncoding RNA FTX regulates cardiomyocyte apoptosis by targeting miR-29b-1-5p and Bcl2l2.
SIRT4 accelerates Ang II-induced pathological cardiac hypertrophy by inhibiting manganese superoxide dismutase activity.
miR-22 regulates starvation-induced autophagy and apoptosis in cardiomyocytes by targeting p38α.
A circular RNA protects the heart from pathological hypertrophy and heart failure by targeting miR-223.
NMNAT3 is involved in the protective effect of SIRT3 in Ang II-induced cardiac hypertrophy.