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Autism-associated Dyrk1a truncation mutants impair neuronal dendritic and spine growth and interfere with postnatal cortical development.
Dang, T; Duan, W Y; Yu, B; Tong, D L; Cheng, C; Zhang, Y F; Wu, W; Ye, K; Zhang, W X; Wu, M; Wu, B B; An, Y; Qiu, Z L; Wu, B L.
Affiliation
  • Dang T; Children's Hospital of Fudan University and Institutes of Biomedical Sciences of Shanghai Medical College, Fudan University, Shanghai, China.
  • Duan WY; Exome Sequencing Collaboration at Boston Children's Hospital and Institute of Biomedical Sciences, Shanghai Medical College of Fudan University, Shanghai, China.
  • Yu B; Institute of Neuroscience, CAS Key Laboratory of Primate Neurobiology, State Key Laboratory of Neuroscience, CAS Center for Excellence in Brain Science and Intelligence Technology, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Shanghai, China.
  • Tong DL; Institute of Neuroscience, CAS Key Laboratory of Primate Neurobiology, State Key Laboratory of Neuroscience, CAS Center for Excellence in Brain Science and Intelligence Technology, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Shanghai, China.
  • Cheng C; Institute of Neuroscience, CAS Key Laboratory of Primate Neurobiology, State Key Laboratory of Neuroscience, CAS Center for Excellence in Brain Science and Intelligence Technology, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Shanghai, China.
  • Zhang YF; Institute of Neuroscience, CAS Key Laboratory of Primate Neurobiology, State Key Laboratory of Neuroscience, CAS Center for Excellence in Brain Science and Intelligence Technology, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Shanghai, China.
  • Wu W; Exome Sequencing Collaboration at Boston Children's Hospital and Institute of Biomedical Sciences, Shanghai Medical College of Fudan University, Shanghai, China.
  • Ye K; Exome Sequencing Collaboration at Boston Children's Hospital and Institute of Biomedical Sciences, Shanghai Medical College of Fudan University, Shanghai, China.
  • Zhang WX; Exome Sequencing Collaboration at Boston Children's Hospital and Institute of Biomedical Sciences, Shanghai Medical College of Fudan University, Shanghai, China.
  • Wu M; Children's Hospital of Fudan University and Institutes of Biomedical Sciences of Shanghai Medical College, Fudan University, Shanghai, China.
  • Wu BB; Exome Sequencing Collaboration at Boston Children's Hospital and Institute of Biomedical Sciences, Shanghai Medical College of Fudan University, Shanghai, China.
  • An Y; Children's Hospital of Fudan University and Institutes of Biomedical Sciences of Shanghai Medical College, Fudan University, Shanghai, China.
  • Qiu ZL; Exome Sequencing Collaboration at Boston Children's Hospital and Institute of Biomedical Sciences, Shanghai Medical College of Fudan University, Shanghai, China.
  • Wu BL; Children's Hospital of Fudan University and Institutes of Biomedical Sciences of Shanghai Medical College, Fudan University, Shanghai, China.
Mol Psychiatry ; 23(3): 747-758, 2018 03.
Article in En | MEDLINE | ID: mdl-28167836
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Protein-Tyrosine Kinases / Protein Serine-Threonine Kinases / Autism Spectrum Disorder Type of study: Prognostic_studies / Risk_factors_studies Limits: Animals / Child / Child, preschool / Female / Humans / Male Language: En Journal: Mol Psychiatry Journal subject: BIOLOGIA MOLECULAR / PSIQUIATRIA Year: 2018 Document type: Article Affiliation country: China Country of publication: United kingdom
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Protein-Tyrosine Kinases / Protein Serine-Threonine Kinases / Autism Spectrum Disorder Type of study: Prognostic_studies / Risk_factors_studies Limits: Animals / Child / Child, preschool / Female / Humans / Male Language: En Journal: Mol Psychiatry Journal subject: BIOLOGIA MOLECULAR / PSIQUIATRIA Year: 2018 Document type: Article Affiliation country: China Country of publication: United kingdom