Disparate Postsynaptic Induction Mechanisms Ultimately Converge to Drive the Retrograde Enhancement of Presynaptic Efficacy.

Goel, Pragya; Li, Xiling; Dickman, Dion

Goel, Pragya; Li, Xiling; Dickman, Dion.

Affiliation

Goel P; Department of Neurobiology, University of Southern California, Los Angeles, CA 90089, USA; Graduate Program in Molecular Biology, University of Southern California, Los Angeles, CA 90089, USA.
Li X; Department of Neurobiology, University of Southern California, Los Angeles, CA 90089, USA.
Dickman D; Department of Neurobiology, University of Southern California, Los Angeles, CA 90089, USA. Electronic address: dickman@usc.edu.

Cell Rep ; 21(9): 2339-2347, 2017 Nov 28.

Article in En | MEDLINE | ID: mdl-29186673

Subject(s)

Drosophila Proteins/metabolism; Presynaptic Terminals/physiology; Animals; Calcium-Calmodulin-Dependent Protein Kinase Type 2/metabolism; Drosophila; Excitatory Postsynaptic Potentials/physiology; Neuromuscular Junction/metabolism; Neuromuscular Junction/physiology; Neuronal Plasticity/physiology; Receptors, Glutamate/metabolism; Signal Transduction/physiology; Synapses/metabolism; Synapses/physiology; Synaptic Transmission/physiology

Key words

Drosophila; homeostasis; neuromuscular junction; retrograde signaling; synaptic plasticity

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Presynaptic Terminals / Drosophila Proteins Limits: Animals Language: En Journal: Cell Rep Year: 2017 Document type: Article Affiliation country: United States Country of publication: United States

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