Detalhe da pesquisa
1.
Gasdermin D is the only Gasdermin that provides protection against acute Salmonella gut infection in mice.
Proc Natl Acad Sci U S A;
120(48): e2315503120, 2023 Nov 28.
Artigo
em Inglês
| MEDLINE
| ID: mdl-37988464
2.
Intraluminal neutrophils limit epithelium damage by reducing pathogen assault on intestinal epithelial cells during Salmonella gut infection.
PLoS Pathog;
19(6): e1011235, 2023 06.
Artigo
em Inglês
| MEDLINE
| ID: mdl-37384776
3.
The Salmonellaâ Typhimurium effector protein SopE transiently localizes to the early SCV and contributes to intracellular replication.
Cell Microbiol;
16(12): 1723-35, 2014 Dec.
Artigo
em Inglês
| MEDLINE
| ID: mdl-25052734
4.
Gut inflammation can boost horizontal gene transfer between pathogenic and commensal Enterobacteriaceae.
Proc Natl Acad Sci U S A;
109(4): 1269-74, 2012 Jan 24.
Artigo
em Inglês
| MEDLINE
| ID: mdl-22232693
5.
Peroral ciprofloxacin therapy impairs the generation of a protective immune response in a mouse model for Salmonella enterica serovar Typhimurium diarrhea, while parenteral ceftriaxone therapy does not.
Antimicrob Agents Chemother;
56(5): 2295-304, 2012 May.
Artigo
em Inglês
| MEDLINE
| ID: mdl-22354292
6.
Intercrypt sentinel macrophages tune antibacterial NF-κB responses in gut epithelial cells via TNF.
J Exp Med;
218(11)2021 11 01.
Artigo
em Inglês
| MEDLINE
| ID: mdl-34529751
7.
Absence of poly(ADP-ribose) polymerase 1 delays the onset of Salmonella enterica serovar Typhimurium-induced gut inflammation.
Infect Immun;
78(8): 3420-31, 2010 Aug.
Artigo
em Inglês
| MEDLINE
| ID: mdl-20515923
8.
Salmonella enterica serovar typhimurium exploits inflammation to compete with the intestinal microbiota.
PLoS Biol;
5(10): 2177-89, 2007 Oct.
Artigo
em Inglês
| MEDLINE
| ID: mdl-17760501
9.
Motility allows S. Typhimurium to benefit from the mucosal defence.
Cell Microbiol;
10(5): 1166-80, 2008 May.
Artigo
em Inglês
| MEDLINE
| ID: mdl-18241212
10.
Salmonella Typhimurium strain ATCC14028 requires H2-hydrogenases for growth in the gut, but not at systemic sites.
PLoS One;
9(10): e110187, 2014.
Artigo
em Inglês
| MEDLINE
| ID: mdl-25303479
11.
Stromal IFN-γR-signaling modulates goblet cell function during Salmonella Typhimurium infection.
PLoS One;
6(7): e22459, 2011.
Artigo
em Inglês
| MEDLINE
| ID: mdl-21829463
12.
IL-17A/F-signaling does not contribute to the initial phase of mucosal inflammation triggered by S. Typhimurium.
PLoS One;
5(11): e13804, 2010 Nov 23.
Artigo
em Inglês
| MEDLINE
| ID: mdl-21124903
13.
Bacterial colitis increases susceptibility to oral prion disease.
J Infect Dis;
199(2): 243-52, 2009 Jan 15.
Artigo
em Inglês
| MEDLINE
| ID: mdl-19072552
14.
Microbe sampling by mucosal dendritic cells is a discrete, MyD88-independent step in DeltainvG S. Typhimurium colitis.
J Exp Med;
205(2): 437-50, 2008 Feb 18.
Artigo
em Inglês
| MEDLINE
| ID: mdl-18268033
15.
Chronic Salmonella enterica serovar Typhimurium-induced colitis and cholangitis in streptomycin-pretreated Nramp1+/+ mice.
Infect Immun;
74(9): 5047-57, 2006 Sep.
Artigo
em Inglês
| MEDLINE
| ID: mdl-16926396
16.
The Salmonella pathogenicity island (SPI)-2 and SPI-1 type III secretion systems allow Salmonella serovar typhimurium to trigger colitis via MyD88-dependent and MyD88-independent mechanisms.
J Immunol;
174(3): 1675-85, 2005 Feb 01.
Artigo
em Inglês
| MEDLINE
| ID: mdl-15661931
17.
Role of the Salmonella pathogenicity island 1 effector proteins SipA, SopB, SopE, and SopE2 in Salmonella enterica subspecies 1 serovar Typhimurium colitis in streptomycin-pretreated mice.
Infect Immun;
72(2): 795-809, 2004 Feb.
Artigo
em Inglês
| MEDLINE
| ID: mdl-14742523
18.
Pretreatment of mice with streptomycin provides a Salmonella enterica serovar Typhimurium colitis model that allows analysis of both pathogen and host.
Infect Immun;
71(5): 2839-58, 2003 May.
Artigo
em Inglês
| MEDLINE
| ID: mdl-12704158