Detalhe da pesquisa
1.
Quantitative Proteomics Analysis of Ischemia/Reperfusion Injury-Modulated Proteins in Cardiac Microvascular Endothelial Cells and the Protective Role of Tongxinluo.
Cell Physiol Biochem;
41(4): 1503-1518, 2017.
Artigo
em Inglês
| MEDLINE
| ID: mdl-28334711
2.
Globular Adiponectin Inhibits the Apoptosis of Mesenchymal Stem Cells Induced by Hypoxia and Serum Deprivation via the AdipoR1-Mediated Pathway.
Cell Physiol Biochem;
38(3): 909-25, 2016.
Artigo
em Inglês
| MEDLINE
| ID: mdl-26919192
3.
Interleukin-5-induced eosinophil population improves cardiac function after myocardial infarction.
Cardiovasc Res;
118(9): 2165-2178, 2022 07 20.
Artigo
em Inglês
| MEDLINE
| ID: mdl-34259869
4.
Strengthening effects of bone marrow mononuclear cells with intensive atorvastatin in acute myocardial infarction.
Open Heart;
7(1)2020 05.
Artigo
em Inglês
| MEDLINE
| ID: mdl-32393654
5.
Optimization of Timing and Times for Administration of Atorvastatin-Pretreated Mesenchymal Stem Cells in a Preclinical Model of Acute Myocardial Infarction.
Stem Cells Transl Med;
8(10): 1068-1083, 2019 10.
Artigo
em Inglês
| MEDLINE
| ID: mdl-31245934
6.
Combined therapy with atorvastatin and atorvastatin-pretreated mesenchymal stem cells enhances cardiac performance after acute myocardial infarction by activating SDF-1/CXCR4 axis.
Am J Transl Res;
11(7): 4214-4231, 2019.
Artigo
em Inglês
| MEDLINE
| ID: mdl-31396330
7.
Transplantation efficacy of autologous bone marrow mesenchymal stem cells combined with atorvastatin for acute myocardial infarction (TEAM-AMI): rationale and design of a randomized, double-blind, placebo-controlled, multi-center, Phase II TEAM-AMI trial.
Regen Med;
14(12): 1077-1087, 2019 12.
Artigo
em Inglês
| MEDLINE
| ID: mdl-31829095
8.
Inhibition of miR-128-3p by Tongxinluo Protects Human Cardiomyocytes from Ischemia/reperfusion Injury via Upregulation of p70s6k1/p-p70s6k1.
Front Pharmacol;
8: 775, 2017.
Artigo
em Inglês
| MEDLINE
| ID: mdl-29163161
9.
AMPK-mediated cardioprotection of atorvastatin relates to the reduction of apoptosis and activation of autophagy in infarcted rat hearts.
Am J Transl Res;
8(10): 4160-4171, 2016.
Artigo
em Inglês
| MEDLINE
| ID: mdl-27830000