Detalhe da pesquisa
1.
Cardiac Oxidative Signaling and Physiological Hypertrophy in the Na/K-ATPase α1s/sα2s/s Mouse Model of High Affinity for Cardiotonic Steroids.
Int J Mol Sci;
22(7)2021 Mar 27.
Artigo
em Inglês
| MEDLINE
| ID: mdl-33801629
2.
The myeloid transcription factor KLF2 regulates the host response to polymicrobial infection and endotoxic shock.
Immunity;
34(5): 715-28, 2011 May 27.
Artigo
em Inglês
| MEDLINE
| ID: mdl-21565532
3.
Isoform-specific role of Na/K-ATPase α1 in skeletal muscle.
Am J Physiol Endocrinol Metab;
314(6): E620-E629, 2018 06 01.
Artigo
em Inglês
| MEDLINE
| ID: mdl-29438630
4.
K⺠and Rb⺠Affinities of the Na,K-ATPase α1 and α2 Isozymes: An Application of ICP-MS for Quantification of Na⺠Pump Kinetics in Myofibers.
Int J Mol Sci;
19(9)2018 Sep 12.
Artigo
em Inglês
| MEDLINE
| ID: mdl-30213059
5.
Pivotal role of α2 Na+ pumps and their high affinity ouabain binding site in cardiovascular health and disease.
J Physiol;
594(21): 6079-6103, 2016 11 01.
Artigo
em Inglês
| MEDLINE
| ID: mdl-27350568
6.
Phospholemman is not required for the acute stimulation of Naâº-Kâº-ATPase α2-activity during skeletal muscle fatigue.
Am J Physiol Cell Physiol;
309(12): C813-22, 2015 Dec 15.
Artigo
em Inglês
| MEDLINE
| ID: mdl-26468207
7.
Na+/Ca2+ exchange and Na+/K+-ATPase in the heart.
J Physiol;
593(6): 1361-82, 2015 Mar 15.
Artigo
em Inglês
| MEDLINE
| ID: mdl-25772291
8.
Reduced levels of microRNAs miR-124a and miR-150 are associated with increased proinflammatory mediator expression in Krüppel-like factor 2 (KLF2)-deficient macrophages.
J Biol Chem;
289(45): 31638-46, 2014 Nov 07.
Artigo
em Inglês
| MEDLINE
| ID: mdl-25248747
9.
Tissue-specific role of the Na,K-ATPase α2 isozyme in skeletal muscle.
J Biol Chem;
288(2): 1226-37, 2013 Jan 11.
Artigo
em Inglês
| MEDLINE
| ID: mdl-23192345
10.
Myeloid-specific Krüppel-like factor 2 inactivation increases macrophage and neutrophil adhesion and promotes atherosclerosis.
Circ Res;
110(10): 1294-302, 2012 May 11.
Artigo
em Inglês
| MEDLINE
| ID: mdl-22474254
11.
The physiological significance of the cardiotonic steroid/ouabain-binding site of the Na,K-ATPase.
Annu Rev Physiol;
72: 395-412, 2010.
Artigo
em Inglês
| MEDLINE
| ID: mdl-20148682
12.
A myeloid hypoxia-inducible factor 1α-Krüppel-like factor 2 pathway regulates gram-positive endotoxin-mediated sepsis.
J Biol Chem;
287(2): 1448-57, 2012 Jan 06.
Artigo
em Inglês
| MEDLINE
| ID: mdl-22110137
13.
Reduction of Na/K-ATPase potentiates marinobufagenin-induced cardiac dysfunction and myocyte apoptosis.
J Biol Chem;
287(20): 16390-8, 2012 May 11.
Artigo
em Inglês
| MEDLINE
| ID: mdl-22451662
14.
Knockout of the Na,K-ATPase α2-isoform in cardiac myocytes delays pressure overload-induced cardiac dysfunction.
Am J Physiol Heart Circ Physiol;
304(8): H1147-58, 2013 Apr 15.
Artigo
em Inglês
| MEDLINE
| ID: mdl-23436327
15.
Histone deacetylase 9 is a negative regulator of adipogenic differentiation.
J Biol Chem;
286(31): 27836-47, 2011 Aug 05.
Artigo
em Inglês
| MEDLINE
| ID: mdl-21680747
16.
Kruppel-like factor 2 regulates thymocyte and T-cell migration.
Nature;
442(7100): 299-302, 2006 Jul 20.
Artigo
em Inglês
| MEDLINE
| ID: mdl-16855590
17.
Renovascular hypertension using a modified two-kidney, one-clip approach in mice is not dependent on the α1 or α2 Na-K-ATPase ouabain-binding site.
Am J Physiol Renal Physiol;
301(3): F615-21, 2011 Sep.
Artigo
em Inglês
| MEDLINE
| ID: mdl-21632957
18.
Mice expressing ouabain-sensitive α1-Na,K-ATPase have increased susceptibility to pressure overload-induced cardiac hypertrophy.
Am J Physiol Heart Circ Physiol;
300(1): H347-55, 2011 Jan.
Artigo
em Inglês
| MEDLINE
| ID: mdl-20952666
19.
Hypertension from chronic central sodium chloride in mice is mediated by the ouabain-binding site on the Na,K-ATPase α2-isoform.
Am J Physiol Heart Circ Physiol;
301(5): H2147-53, 2011 Nov.
Artigo
em Inglês
| MEDLINE
| ID: mdl-21856907
20.
Knockout of the Na,K-ATPase α2-isoform in the cardiovascular system does not alter basal blood pressure but prevents ACTH-induced hypertension.
Am J Physiol Heart Circ Physiol;
301(4): H1396-404, 2011 Oct.
Artigo
em Inglês
| MEDLINE
| ID: mdl-21856916