Detalhe da pesquisa
1.
EGFR/IGF1R Signaling Modulates Relaxation in Hypertrophic Cardiomyopathy.
Circ Res;
133(5): 387-399, 2023 08 18.
Artigo
em Inglês
| MEDLINE
| ID: mdl-37477020
2.
Depletion of Vasohibin 1 Speeds Contraction and Relaxation in Failing Human Cardiomyocytes.
Circ Res;
127(2): e14-e27, 2020 07 03.
Artigo
em Inglês
| MEDLINE
| ID: mdl-32272864
3.
Mechanistic role of the CREB-regulated transcription coactivator 1 in cardiac hypertrophy.
J Mol Cell Cardiol;
127: 31-43, 2019 02.
Artigo
em Inglês
| MEDLINE
| ID: mdl-30521840
4.
Myoarchitectural disarray of hypertrophic cardiomyopathy begins pre-birth.
J Anat;
235(5): 962-976, 2019 11.
Artigo
em Inglês
| MEDLINE
| ID: mdl-31347708
5.
Knock-out of nexilin in mice leads to dilated cardiomyopathy and endomyocardial fibroelastosis.
Basic Res Cardiol;
111(1): 6, 2016 Jan.
Artigo
em Inglês
| MEDLINE
| ID: mdl-26659360
6.
The E3 ubiquitin ligase Asb2ß is downregulated in a mouse model of hypertrophic cardiomyopathy and targets desmin for proteasomal degradation.
J Mol Cell Cardiol;
87: 214-24, 2015 Oct.
Artigo
em Inglês
| MEDLINE
| ID: mdl-26343497
7.
Sexual dimorphic response to exercise in hypertrophic cardiomyopathy-associated MYBPC3-targeted knock-in mice.
Pflugers Arch;
467(6): 1303-17, 2015 Jun.
Artigo
em Inglês
| MEDLINE
| ID: mdl-25010737
8.
Heterozygous LmnadelK32 mice develop dilated cardiomyopathy through a combined pathomechanism of haploinsufficiency and peptide toxicity.
Hum Mol Genet;
22(15): 3152-64, 2013 Aug 01.
Artigo
em Inglês
| MEDLINE
| ID: mdl-23575224
9.
Ubiquitin-proteasome system and hereditary cardiomyopathies.
J Mol Cell Cardiol;
71: 25-31, 2014 Jun.
Artigo
em Inglês
| MEDLINE
| ID: mdl-24380728
10.
DelK32-lamin A/C has abnormal location and induces incomplete tissue maturation and severe metabolic defects leading to premature death.
Hum Mol Genet;
21(5): 1037-48, 2012 Mar 01.
Artigo
em Inglês
| MEDLINE
| ID: mdl-22090424
11.
Chronic activation of tubulin tyrosination in HCM mice and human iPSC-engineered heart tissues improves heart function.
bioRxiv;
2024 Feb 07.
Artigo
em Inglês
| MEDLINE
| ID: mdl-37292763
12.
Contractile abnormalities and altered drug response in engineered heart tissue from Mybpc3-targeted knock-in mice.
J Mol Cell Cardiol;
63: 189-98, 2013 Oct.
Artigo
em Inglês
| MEDLINE
| ID: mdl-23896226
13.
The expression of podocyte-specific proteins in parietal epithelial cells is regulated by protein degradation.
Kidney Int;
84(3): 532-44, 2013 Sep.
Artigo
em Inglês
| MEDLINE
| ID: mdl-23615505
14.
Defective proteolytic systems in Mybpc3-targeted mice with cardiac hypertrophy.
Basic Res Cardiol;
107(1): 235, 2012 Jan.
Artigo
em Inglês
| MEDLINE
| ID: mdl-22189562
15.
How do MYBPC3 mutations cause hypertrophic cardiomyopathy?
J Muscle Res Cell Motil;
33(1): 75-80, 2012 May.
Artigo
em Inglês
| MEDLINE
| ID: mdl-22057632
16.
Adrenergic stress reveals septal hypertrophy and proteasome impairment in heterozygous Mybpc3-targeted knock-in mice.
J Muscle Res Cell Motil;
33(1): 5-15, 2012 May.
Artigo
em Inglês
| MEDLINE
| ID: mdl-22076249
17.
CMYA5 is a novel interaction partner of FHL2 in cardiac myocytes.
FEBS J;
289(15): 4622-4645, 2022 08.
Artigo
em Inglês
| MEDLINE
| ID: mdl-35176204
18.
ACTN2 Mutant Causes Proteopathy in Human iPSC-Derived Cardiomyocytes.
Cells;
11(17)2022 09 02.
Artigo
em Inglês
| MEDLINE
| ID: mdl-36078153
19.
Cardiac myosin-binding protein C in hypertrophic cardiomyopathy: mechanisms and therapeutic opportunities.
J Mol Cell Cardiol;
50(4): 613-20, 2011 Apr.
Artigo
em Inglês
| MEDLINE
| ID: mdl-21291890
20.
The ubiquitin-proteasome system in cardiomyopathies.
Curr Opin Cardiol;
26(3): 190-5, 2011 May.
Artigo
em Inglês
| MEDLINE
| ID: mdl-21415726