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HMG-CoA reductase promotes protein prenylation and therefore is indispensible for T-cell survival.
Lacher, Sonja M; Bruttger, Julia; Kalt, Bettina; Berthelet, Jean; Rajalingam, Krishnaraj; Wörtge, Simone; Waisman, Ari.
Afiliação
  • Lacher SM; Institute for Molecular Medicine, University Medical Center of the Johannes Gutenberg-University Mainz, Obere Zahlbacher Str. 67, Mainz 55131, Germany.
  • Bruttger J; Institute for Molecular Medicine, University Medical Center of the Johannes Gutenberg-University Mainz, Obere Zahlbacher Str. 67, Mainz 55131, Germany.
  • Kalt B; Institute for Molecular Medicine, University Medical Center of the Johannes Gutenberg-University Mainz, Obere Zahlbacher Str. 67, Mainz 55131, Germany.
  • Berthelet J; Molecular Signaling Unit, FZI, Institute of Immunology, University Medical Center of the Johannes Gutenberg-University Mainz, Langenbeck Str. 1, Mainz 55131, Germany.
  • Rajalingam K; Molecular Signaling Unit, FZI, Institute of Immunology, University Medical Center of the Johannes Gutenberg-University Mainz, Langenbeck Str. 1, Mainz 55131, Germany.
  • Wörtge S; Institute for Molecular Medicine, University Medical Center of the Johannes Gutenberg-University Mainz, Obere Zahlbacher Str. 67, Mainz 55131, Germany.
  • Waisman A; Institute for Molecular Medicine, University Medical Center of the Johannes Gutenberg-University Mainz, Obere Zahlbacher Str. 67, Mainz 55131, Germany.
Cell Death Dis ; 8(5): e2824, 2017 05 25.
Article em En | MEDLINE | ID: mdl-28542128
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Linfócitos T / Prenilação de Proteína / Hidroximetilglutaril-CoA Redutases Limite: Animals Idioma: En Revista: Cell Death Dis Ano de publicação: 2017 Tipo de documento: Article País de afiliação: Alemanha
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Linfócitos T / Prenilação de Proteína / Hidroximetilglutaril-CoA Redutases Limite: Animals Idioma: En Revista: Cell Death Dis Ano de publicação: 2017 Tipo de documento: Article País de afiliação: Alemanha