IL-17: an important pathogenic factor in endometriosis.

Shi, Jia-Lu; Zheng, Zi-Meng; Chen, Min; Shen, Hui-Hui; Li, Ming-Qing; Shao, Jun

Shi, Jia-Lu; Zheng, Zi-Meng; Chen, Min; Shen, Hui-Hui; Li, Ming-Qing; Shao, Jun.

Afiliação

Shi JL; Laboratory for Reproductive Immunology, Hospital of Obstetrics and Gynecology, Fudan University, Shanghai 200080, People's Republic of China.
Zheng ZM; Laboratory for Reproductive Immunology, Hospital of Obstetrics and Gynecology, Fudan University, Shanghai 200080, People's Republic of China.
Chen M; NHC Key Lab of Reproduction Regulation (Shanghai Institute for Biomedical and Pharmaceutical Technologies), Hospital of Obstetrics and Gynecology, Fudan University, Shanghai 200080, People's Republic of China.
Shen HH; Laboratory for Reproductive Immunology, Hospital of Obstetrics and Gynecology, Fudan University, Shanghai 200080, People's Republic of China.
Li MQ; Laboratory for Reproductive Immunology, Hospital of Obstetrics and Gynecology, Fudan University, Shanghai 200080, People's Republic of China.
Shao J; Laboratory for Reproductive Immunology, Hospital of Obstetrics and Gynecology, Fudan University, Shanghai 200080, People's Republic of China.

Int J Med Sci ; 19(4): 769-778, 2022.

Article em En | MEDLINE | ID: mdl-35582411

Assuntos

Endometriose; Interleucina-17; Líquido Ascítico/metabolismo; Endometriose/etiologia; Feminino; Humanos; Interleucina-17/genética; Interleucina-17/metabolismo; Células Th17/metabolismo

Palavras-chave

IL-17; Th17 cell; cytokine; endometriosis; inflammatory

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Interleucina-17 / Endometriose Tipo de estudo: Etiology_studies Limite: Female / Humans Idioma: En Revista: Int J Med Sci Assunto da revista: MEDICINA Ano de publicação: 2022 Tipo de documento: Article

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