MRI findings in adolescent patients with acute traumatic knee hemarthrosis.

BACKGROUND: Physical examination may be inconclusive in adolescents presenting with an acute traumatic knee effusion because of pain and guarding. The purpose of this study was to describe the magnetic resonance imaging (MRI) findings in adolescents with traumatic knee effusions and to compare injuries based on age, sex, and physeal maturity. METHODS: All MRIs using a knee trauma protocol performed at our institution over a 2-year period were evaluated. One hundred thirty-one patients between the ages of 10 to 18 years of age with a clinical history of acute knee trauma and an effusion confirmed on MRI met our study inclusion criteria. They were divided into 2 age groups: 10 to 14 and 15 to 18 years old. Pathology was confirmed using clinical history, MRI, and any available surgical reports. RESULTS: Of the 131 patients with an acute knee effusion, there were 59 patients in the younger group (10 to 14 y old) and 72 patients in the older group (15 to 18 y old). In the younger group, patellar dislocations (36%), anterior cruciate ligament (ACL) tears (22%), and isolated meniscus tears (15%) were the most common injuries. In the older group, ACL tears (40%), patellar dislocations (28%), and isolated meniscus tears (13%) were the most common injuries. ACL injuries represented 28% of injuries in males and 38% of injuries in females, whereas patellar dislocations represented 28% of injuries in males and 37% of injuries in females. There was a trend toward adolescents with active growth plates sustaining more patellar dislocations and adolescents with closed growth plates sustaining more ACL injuries. Forty-one percent of patients in this study underwent surgery. CONCLUSIONS: Patellar dislocation is a common injury in children who present with a traumatic knee effusion, especially in young adolescents and females. Adolescents presenting with a traumatic knee effusion should undergo MRI because of the high rate of positive findings missed by physical examination and plain radiographs that may warrant surgical repair or reconstruction. LEVEL OF EVIDENCE: Level III.

The prevalence of hereditary neuropathy with liability to pressure palsies in patients with multiple surgically treated entrapment neuropathies.

PURPOSE: Hereditary neuropathy with liability to pressure palsies (HNPP) is an autosomal-dominant peripheral neuropathy that results from deletion of a 1.5-Megabase pair (Mb) segment of the short arm (p) of chromosome 17. Hereditary neuropathy with liability to pressure palsies increases susceptibility of peripheral nerves to pressure and trauma and can be associated with symptoms at multiple anatomic entrapment sites. Many patients present with multiple upper-extremity entrapment neuropathies and the etiology is uncertain. We hypothesized that some of these patients have an underlying hereditary neuropathy. The purpose of this study was to determine the prevalence of HNPP in patients with multiple surgically treated upper-extremity entrapment neuropathies. METHODS: The inclusion criterion for the study was history of more than 1 carpal tunnel release and/or ulnar nerve transposition. The exclusion criteria were history of diabetes or history of Charcot-Marie-Tooth neuropathy. Fifty-nine patients were in the study group. Two patients known to have the 17p11.2 deletion were used as controls. Genomic DNA was extracted from peripheral blood. Each sample was genotyped using polymerase chain reaction (PCR) amplification with short tandem repeat polymorphism markers within the 1.5-Mb region of 17p deleted in HNPP. Markers were scored as homozygous or heterozygous after resolution by polyacrylamide gel electrophoresis and silver staining. RESULTS: The 2 control patients were homozygous for 11 markers. None of the 59 study patients were homozygous for all markers tested in the deleted region. No study patient had the 17p deletion diagnostic for HNPP. Based on the sample size of 59 patients the 95% confidence interval for the prevalence of the 17p11.2 deletion in this population is 0% to 5%. CONCLUSIONS: We found no evidence for an association between HNPP and patients who have multiple surgical releases for upper-extremity entrapment neuropathies.

Results of an accelerated Ponseti protocol for clubfoot.

The Ponseti method has demonstrated excellent results when performed at weekly intervals, but it is not known whether correction can be accomplished in a shorter amount of time. This study evaluated the success in correction in relation to time between casts (5 or 7 days). The authors retrospectively reviewed 230 patients (319 clubfeet). One hundred sixty-five patients (72%) had undergone previous nonsurgical treatment elsewhere. Patients were assigned to 5 or 7 days based solely on geography. Ninety percent of patients required five or fewer casts for correction, and there was no difference between groups (P = 0.85). Average time from first cast to Achilles tenotomy was 16 days for the 5-day group and 24 days for the 7-day group (P = 0.001). Three patients (1.3%) required corrective surgery and there were 36 relapses (P = 0.4). In conclusion, the Ponseti method is very effective and the deformity can be corrected in a relatively short time.