ß-Alanine Plus Sodium Bicarbonate Co-Supplementation Does Not Decrease Neuromuscular Fatigue in Swimming.

This study aimed to investigate the effects of chronic ß-alanine (ßA) plus acute sodium bicarbonate (SB) co-supplementation on neuromuscular fatigue during high-intensity intermittent efforts in swimming. Eleven regional and national competitive-level young swimmers performed a neuromuscular fatigue assessment before and immediately after two 20 × 25-m front crawl maximal efforts every 90 s, performed at pre- and post-4-week co-supplementation. Neuromuscular fatigue was evaluated by percutaneous electrical stimuli through the twitch interpolation technique on the triceps brachii and quadriceps femoris. Performance was defined by the mean time of the 20 efforts and blood samples to lactate concentrations were collected every four efforts. Participants supplemented 3.2-6.4 g·day-1 of chronic ßA or placebo (PL) during four weeks, and acute 0.3 g·kg-1 of SB or PL 60 min before the second assessment (allowing ßA+SB and PL+PL groups). No statistical changes were found in neuromuscular fatigue of triceps brachii. In the quadriceps femoris, a main effect of time was found in potentiated twitch delta values in pooled groups, showing a statistical increase of 19.01% after four weeks (Δ = 13.05 [0.35-25.75] N; p = 0.044), without time × group interactions. No statistical difference was found in the swimming performance. Blood lactate increased by 25.06% only in the ßA+SB group (Δ = 6.40 [4.62-8.18] mM; p Bonf < 0.001) after the supplementation period. In conclusion, 4-week ßA and SB co-supplementation were not able to reduce neuromuscular fatigue levels and improve performance in highintensity intermittent efforts, but statistically increased blood lactate levels.

Four-week experimental plus 1-week taper period using live high train low does not alter muscle glycogen content.

This study aimed to investigate the effects of a 4-week live high train low (LHTL; FiO2 ~ 13.5%), intervention, followed by a tapering phase, on muscle glycogen concentration. Fourteen physically active males (28 ± 6 years, 81.6 ± 15.4 kg, 179 ± 5.2 cm) were divided into a control group (CON; n = 5), and the group that performed the LHTL, which was exposed to hypoxia (LHTL; n = 9). The subjects trained using a one-legged knee extension exercise, which enabled four experimental conditions: leg training in hypoxia (TLHYP); leg control in hypoxia (CLHYP, n = 9); leg trained in normoxia (TLNOR, n = 5), and leg control in normoxia (CLNOR, n = 5). All participants performed 18 training sessions lasting between 20 and 45 min [80-200% of intensity corresponding to the time to exhaustion (TTE) reached in the graded exercise test]. Additionally, participants spent approximately 10 h day-1 in either a normobaric hypoxic environment (14.5% FiO2; ~ 3000 m) or a control condition (i.e., staying in similar tents on ~ 530 m). Thereafter, participants underwent a taper protocol consisting of six additional training sessions with a reduced training load. SpO2 was lower, and the hypoxic dose was higher in LHTL compared to CON (p < 0.001). After 4 weeks, glycogen had increased significantly only in the TLNOR and TLHYP groups and remained elevated after the taper (p < 0.016). Time to exhaustion in the LHTL increased after both the 4-week training period and the taper compared to the baseline (p < 0.001). Although the 4-week training promoted substantial increases in muscle glycogen content, TTE increased in LHTL condition.

Effects of an experimental taper period on male and female swimmers.

BACKGROUND: This study investigated the possible influence of the gender on the responses of swimmers during a taper period (TP). METHODS: Ten males (19±3 years and 73.5±7.8 kg) and ten females (17±2 years and 54.7±7.2 kg) swimmers were submitted to a 12-week training, followed by three weeks of the TP. Before and after the TP we evaluated the performance at 100 m freestyle, stroke parameters and lactacidemic responses; lactate minimum intensity (LMI) and stroke parameters associated with LMI and the propulsive force in tethered swimming. TP consisted of 14 sessions with mean volume 2253±1213 m/session at an intensity below than the LMI, 1730±327 m/session at an intensity near the LMI and 1530±1019 m/session at an intensity above the LMI. RESULTS: Significant effects of the genders were observed for LMI and stroke parameters (P<0.001 and η2>0.52 [large]) and propulsive force (P=0.001; η2=0.59 [large]). However, no significant effects of the TP were identified in the performance of the 100 m freestyle (P=0.66; η2=0.006 [small]), propulsive force (P>0.63; η2<0.006 [small]), aerobic parameters (LMI: P=0.32 and η2=0.03 [small]) and mechanical parameters (P>0.23; η2=0.01 [small]). Nonetheless, the peak blood lactate concentrations were improved after TP (P=0.014; η2=0.16 [large]), without significant interactions (P=0.38; η2=0.02 [small]), as well as the mechanical parameters during maximum 100 m freestyle (P<0.04 and η2>0.10 [medium]). CONCLUSIONS: Hence, men and women presenting significantly different values in the age group studied, the responses observed after the TP investigated were the same independent of gender.