1.
Mol Cancer Res
; 21(1): 76-85, 2023 01 03.
Artigo
em Inglês
| MEDLINE
| ID: mdl-36162957
RESUMO
IMPLICATIONS: Considering the importance of GSTA4 in controlling IFNγ responsiveness and the metastatic potential of other melanoma cells, our results highlight a novel mechanism whereby cancer cells escape from host immunity and gain metastatic ability by acquiring resistance to oxidative stress responses through the upregulation of GSTA4.