Mentally retarded persons as parents: prevalence and the situation of their children.

This paper describes a research program on mentally retarded parents and their children. For this nation-wide study, two structured questionnaires were sent to all municipalities in Norway. Both questionnaires were sent to public health nurses and were followed-up by a structured telephone interview based on the questionnaires. The primary aim of the study was to survey the number of children born to mentally retarded parents. In addition, the children's needs and functional abilities were to be assessed. Twenty-three mentally retarded persons had given birth in the course of the past twelve months. A total of 126 children with mentally retarded parents were identified, with an incidence of 27 children per year, and a prevalence of approximately 430 children under 16 years of age in a population of 4 mill, people with a mean of 1.05 child per family. About 43% of the children of mentally retarded parents appeared to have learning difficulties. Forty percent of the children suffered from failures of care. Between 25% and 68% of the children with learning problems had poorly developed motor or language abilities, sense modalities or psycho-social status.

Exploration and avoidance in rats with lesions in amygdala and piriform cortex.

Lesions localized to specific areas of the amygdala and overlying cortex in rats produced differential effects in several behavioral tasks. Three different types of lesions were tested: central, basolateral, and cortex lateral to the amygdala. Lesions restricted to the central nucleus produced increased activity on all parameters studied in an open-field test, but the other two groups were not changed. In one-way active avoidance all three groups with lesions showed deficits. The most pronounced change was observed in the central group. All groups showed the same degree of retention loss, but in forced extinction of one-way active avoidance after retraining, the cortical and basolateral groups were most defective. A fear-reduction hypothesis is proposed for the central lesion. The basolateral and cortical areas may be more specifically involved in passive avoidance behavior.