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Knock out of S1P3 receptor signaling attenuates inflammation and fibrosis in bleomycin-induced lung injury mice model.
Murakami, Ken; Kohno, Masataka; Kadoya, Masatoshi; Nagahara, Hidetake; Fujii, Wataru; Seno, Takahiro; Yamamoto, Aihiro; Oda, Ryo; Fujiwara, Hiroyoshi; Kubo, Toshikazu; Morita, Satoshi; Nakada, Hiroshi; Hla, Timothy; Kawahito, Yutaka.
Afiliación
  • Murakami K; Inflammation and Immunology, Graduate school of Medical Science, Kyoto Prefectural University of Medicine, Kyoto, Japan.
  • Kohno M; Inflammation and Immunology, Graduate school of Medical Science, Kyoto Prefectural University of Medicine, Kyoto, Japan.
  • Kadoya M; Inflammation and Immunology, Graduate school of Medical Science, Kyoto Prefectural University of Medicine, Kyoto, Japan.
  • Nagahara H; Inflammation and Immunology, Graduate school of Medical Science, Kyoto Prefectural University of Medicine, Kyoto, Japan.
  • Fujii W; Inflammation and Immunology, Graduate school of Medical Science, Kyoto Prefectural University of Medicine, Kyoto, Japan.
  • Seno T; Inflammation and Immunology, Graduate school of Medical Science, Kyoto Prefectural University of Medicine, Kyoto, Japan; Department of Rheumatic Diseases and Joint Function, Kyoto Prefectural University of Medicine, Kyoto, Japan.
  • Yamamoto A; Inflammation and Immunology, Graduate school of Medical Science, Kyoto Prefectural University of Medicine, Kyoto, Japan.
  • Oda R; Department of Orthopedics, Graduate School of Medical Science, Kyoto Prefectural University of Medicine, Kyoto, Japan.
  • Fujiwara H; Department of Orthopedics, Graduate School of Medical Science, Kyoto Prefectural University of Medicine, Kyoto, Japan.
  • Kubo T; Department of Rheumatic Diseases and Joint Function, Kyoto Prefectural University of Medicine, Kyoto, Japan; Department of Orthopedics, Graduate School of Medical Science, Kyoto Prefectural University of Medicine, Kyoto, Japan.
  • Morita S; Department of Biomedical Statistics and Bioinformatics, Kyoto University Graduate School of Medicine, Kyoto, Japan.
  • Nakada H; Department of Molecular Biosciences, Faculty of Life Sciences, Kyoto Sangyo University, Kyoto, Japan.
  • Hla T; Department of Pathology and Laboratory Medicine, Weill Cornell Medical College, New York, New York, United States of America.
  • Kawahito Y; Inflammation and Immunology, Graduate school of Medical Science, Kyoto Prefectural University of Medicine, Kyoto, Japan.
PLoS One ; 9(9): e106792, 2014.
Article en En | MEDLINE | ID: mdl-25198418
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Bleomicina / Fibrosis / Transducción de Señal / Receptores de Lisoesfingolípidos / Inflamación / Pulmón Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: PLoS One Asunto de la revista: CIENCIA / MEDICINA Año: 2014 Tipo del documento: Article País de afiliación: Japón
Texto completo
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XML
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Bleomicina / Fibrosis / Transducción de Señal / Receptores de Lisoesfingolípidos / Inflamación / Pulmón Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: PLoS One Asunto de la revista: CIENCIA / MEDICINA Año: 2014 Tipo del documento: Article País de afiliación: Japón