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HIF2α contributes to antiestrogen resistance via positive bilateral crosstalk with EGFR in breast cancer cells.
Alam, Muhammad Wasi; Persson, Camilla Ulrika; Reinbothe, Susann; Kazi, Julhash U; Rönnstrand, Lars; Wigerup, Caroline; Ditzel, Henrik Jorn; Lykkesfeldt, Anne E; Påhlman, Sven; Jögi, Annika.
Afiliación
  • Alam MW; Department of Laboratory Medicine, Translational Cancer Research, Lund University Cancer Center at Medicon Village, Lund University, Sweden.
  • Persson CU; Department of Laboratory Medicine, Translational Cancer Research, Lund University Cancer Center at Medicon Village, Lund University, Sweden.
  • Reinbothe S; Department of Laboratory Medicine, Translational Cancer Research, Lund University Cancer Center at Medicon Village, Lund University, Sweden.
  • Kazi JU; Department of Laboratory Medicine, Translational Cancer Research, Lund University Cancer Center at Medicon Village, Lund University, Sweden.
  • Rönnstrand L; Department of Laboratory Medicine, Translational Cancer Research, Lund University Cancer Center at Medicon Village, Lund University, Sweden.
  • Wigerup C; Department of Laboratory Medicine, Translational Cancer Research, Lund University Cancer Center at Medicon Village, Lund University, Sweden.
  • Ditzel HJ; Department of Cancer and Inflammation Research, University of Southern Denmark, and Department of Oncology, Odense University Hospital, Odense, Denmark.
  • Lykkesfeldt AE; Breast Cancer Group, Cell Death and Metabolism, Danish Cancer Society Research Center, Copenhagen, Denmark.
  • Påhlman S; Department of Laboratory Medicine, Translational Cancer Research, Lund University Cancer Center at Medicon Village, Lund University, Sweden.
  • Jögi A; Department of Laboratory Medicine, Translational Cancer Research, Lund University Cancer Center at Medicon Village, Lund University, Sweden.
Oncotarget ; 7(10): 11238-50, 2016 Mar 08.
Article en En | MEDLINE | ID: mdl-26849233
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Neoplasias de la Mama / Resistencia a Antineoplásicos / Moduladores de los Receptores de Estrógeno / Factores de Transcripción con Motivo Hélice-Asa-Hélice Básico / Receptores ErbB Límite: Female / Humans Idioma: En Revista: Oncotarget Año: 2016 Tipo del documento: Article País de afiliación: Suecia
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Neoplasias de la Mama / Resistencia a Antineoplásicos / Moduladores de los Receptores de Estrógeno / Factores de Transcripción con Motivo Hélice-Asa-Hélice Básico / Receptores ErbB Límite: Female / Humans Idioma: En Revista: Oncotarget Año: 2016 Tipo del documento: Article País de afiliación: Suecia