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Ginsenoside Rg1 inhibits angiotensin II-induced podocyte autophagy via AMPK/mTOR/PI3K pathway.
Mao, Nan; Tan, Rui-Zhi; Wang, Shao-Qing; Wei, Cong; Shi, Xin-Li; Fan, Jun-Ming; Wang, Li.
Afiliación
  • Mao N; Department of Nephrology, The First Affiliated Hospital of Chengdu Medical College, Chengdu, Sichuan, 610500, China.
  • Tan RZ; Research Center of Combine Traditional Chinese and Western Medicine, Affiliated Traditional Medicine Hospital, Southwest Medical University, Luzhou, Sichuan, 646000, China.
  • Wang SQ; Department of Nephrology, The First Affiliated Hospital of Chengdu Medical College, Chengdu, Sichuan, 610500, China.
  • Wei C; Clinical Laboratory, Affiliated Traditional Medicine Hospital, Southwest Medical University, Luzhou, Sichuan, 646000, China.
  • Shi XL; Department of Pathobiology and Immunology, Hebei University of Chinese Medicine, Hebei, Shijiazhuang, 050200, China.
  • Fan JM; Department of Nephrology, Affiliated Traditional Medicine Hospital, Southwest Medical University, Luzhou, Sichuan, 646000, China.
  • Wang L; Research Center of Combine Traditional Chinese and Western Medicine, Affiliated Traditional Medicine Hospital, Southwest Medical University, Luzhou, Sichuan, 646000, China.
Cell Biol Int ; 40(8): 917-25, 2016 Aug.
Article en En | MEDLINE | ID: mdl-27296076
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Autofagia / Angiotensina II / Fosfatidilinositol 3-Quinasas / Ginsenósidos / Podocitos / Proteínas Quinasas Activadas por AMP / Serina-Treonina Quinasas TOR Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Cell Biol Int Año: 2016 Tipo del documento: Article País de afiliación: China
Texto completo
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Autofagia / Angiotensina II / Fosfatidilinositol 3-Quinasas / Ginsenósidos / Podocitos / Proteínas Quinasas Activadas por AMP / Serina-Treonina Quinasas TOR Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Cell Biol Int Año: 2016 Tipo del documento: Article País de afiliación: China