Endoplasmic Reticulum Stress Promotes iNOS/NO and Influences Inflammation in the Development of Doxorubicin-Induced Cardiomyopathy.

Bagchi, Ashim K; Malik, Akshi; Akolkar, Gauri; Jassal, Davinder S; Singal, Pawan K

Bagchi, Ashim K; Malik, Akshi; Akolkar, Gauri; Jassal, Davinder S; Singal, Pawan K.

Afiliación

Bagchi AK; St. Boniface Hospital Albrechtsen Research Centre, Department of Physiology and Pathophysiology, Institute of Cardiovascular Sciences, University of Manitoba, Winnipeg, MB R2H 2A6, Canada.
Malik A; St. Boniface Hospital Albrechtsen Research Centre, Department of Physiology and Pathophysiology, Institute of Cardiovascular Sciences, University of Manitoba, Winnipeg, MB R2H 2A6, Canada.
Akolkar G; Cardio-Renal Division, Therapeutic Products Directorate, Ottawa, ON K1A 0K9, Canada.
Jassal DS; St. Boniface Hospital Albrechtsen Research Centre, Department of Physiology and Pathophysiology, Institute of Cardiovascular Sciences, University of Manitoba, Winnipeg, MB R2H 2A6, Canada.
Singal PK; Section of Cardiology, Department of Internal Medicine, Rady Faculty of Health Sciences, University of Manitoba, Winnipeg, MB R2H 2A6, Canada.

Antioxidants (Basel) ; 10(12)2021 Nov 26.

Article en En | MEDLINE | ID: mdl-34943000

Palabras clave

Dox-induced cardiomyopathy; ER stress; Toll-like receptor 2; apoptosis; inducible nitric oxide synthase

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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: Antioxidants (Basel) Año: 2021 Tipo del documento: Article País de afiliación: Canadá

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