Methionine restriction forces Epstein-Barr virus out of latency.

Kashyap, Sriraksha Bharadwaj; Mulondo, Racheal; Mullen, Peter J

Kashyap, Sriraksha Bharadwaj; Mulondo, Racheal; Mullen, Peter J.

Afiliación

Kashyap SB; Department of Molecular Microbiology and Immunology, Keck School of Medicine, University of Southern California, Los Angeles, CA 90089, USA.
Mulondo R; Department of Molecular Microbiology and Immunology, Keck School of Medicine, University of Southern California, Los Angeles, CA 90089, USA.
Mullen PJ; Department of Molecular Microbiology and Immunology, Keck School of Medicine, University of Southern California, Los Angeles, CA 90089, USA; Leonard Davis School of Gerontology, University of Southern California, Los Angeles, CA 90089, USA; Norris Comprehensive Cancer Center, Keck School of Medicine, University of Southern California, Los Angeles CA 90089, USA. Electronic address: petermul@usc.edu.

Cell Metab ; 34(9): 1229-1231, 2022 09 06.

Article en En | MEDLINE | ID: mdl-36070678

ABSTRACT

ABSTRACT

EBV gene expression is repressed during viral latency to prevent an immune response, but it is not known how metabolism contributes to this silencing. In this issue of Cell Metabolism, Guo et al. describe how methionine restriction reactivates the expression of EBV genes, offering new therapeutic approaches against EBV-driven diseases.

Asunto(s)

Infecciones por Virus de Epstein-Barr; Herpesvirus Humano 4; Humanos; Metionina; Latencia del Virus

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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Herpesvirus Humano 4 / Infecciones por Virus de Epstein-Barr Límite: Humans Idioma: En Revista: Cell Metab Asunto de la revista: METABOLISMO Año: 2022 Tipo del documento: Article País de afiliación: Estados Unidos

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