CD28-signaling can be partially compensated in CD28-knockout mice but is essential for virus elimination in a murine model of multiple sclerosis.

Hülskötter, Kirsten; Lühder, Fred; Leitzen, Eva; Flügel, Alexander; Baumgärtner, Wolfgang

Hülskötter, Kirsten; Lühder, Fred; Leitzen, Eva; Flügel, Alexander; Baumgärtner, Wolfgang.

Afiliación

Hülskötter K; Department of Pathology, University of Veterinary Medicine Hannover, Foundation, Hannover, Germany.
Lühder F; Institute for Neuroimmunology and Multiple Sclerosis Research (IMSF), University Medical Center Goettingen, Goettingen, Germany.
Leitzen E; Department of Pathology, University of Veterinary Medicine Hannover, Foundation, Hannover, Germany.
Flügel A; Institute for Neuroimmunology and Multiple Sclerosis Research (IMSF), University Medical Center Goettingen, Goettingen, Germany.
Baumgärtner W; Department of Pathology, University of Veterinary Medicine Hannover, Foundation, Hannover, Germany.

Front Immunol ; 14: 1105432, 2023.

Article en En | MEDLINE | ID: mdl-37090733

Asunto(s)

Esclerosis Múltiple; Ratones; Animales; Antígenos CD28/genética; Modelos Animales de Enfermedad; Ratones Noqueados; Ratones Endogámicos C57BL

Palabras clave

Theiler's murine encephalomyelitis virus (TMEV); conditional CD28-knockout; conventional CD28-knockout; immunology & infectious diseases; multiple sclerosis (MS) disease; neuroimmunology and neuropathology

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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Esclerosis Múltiple Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Front Immunol Año: 2023 Tipo del documento: Article País de afiliación: Alemania

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