Mechanisms of action of therapeutic amyloidogenic hexapeptides in amelioration of inflammatory brain disease.

Kurnellas, Michael P; Schartner, Jill M; Fathman, C Garrison; Jagger, Ann; Steinman, Lawrence; Rothbard, Jonathan B

Kurnellas, Michael P; Schartner, Jill M; Fathman, C Garrison; Jagger, Ann; Steinman, Lawrence; Rothbard, Jonathan B.

Afiliação

Kurnellas MP; Department of Neurology and Neurological Sciences and Department of Medicine, Division of Immunology, Stanford University School of Medicine, Stanford, CA 94305.
Schartner JM; Department of Neurology and Neurological Sciences and Department of Medicine, Division of Immunology, Stanford University School of Medicine, Stanford, CA 94305.
Fathman CG; Department of Neurology and Neurological Sciences and Department of Medicine, Division of Immunology, Stanford University School of Medicine, Stanford, CA 94305.
Jagger A; Department of Neurology and Neurological Sciences and Department of Medicine, Division of Immunology, Stanford University School of Medicine, Stanford, CA 94305.
Steinman L; Department of Neurology and Neurological Sciences and Department of Medicine, Division of Immunology, Stanford University School of Medicine, Stanford, CA 94305 steinman@stanford.edu.
Rothbard JB; Department of Neurology and Neurological Sciences and Department of Medicine, Division of Immunology, Stanford University School of Medicine, Stanford, CA 94305 Department of Neurology and Neurological Sciences and Department of Medicine, Division of Immunology, Stanford University School of Medicin

J Exp Med ; 211(9): 1847-56, 2014 Aug 25.

Article em En | MEDLINE | ID: mdl-25073790

Assuntos

Amiloide/imunologia; Amiloide/uso terapêutico; Encefalomielite Autoimune Experimental/imunologia; Encefalomielite Autoimune Experimental/terapia; Fragmentos de Peptídeos/imunologia; Fragmentos de Peptídeos/uso terapêutico; Transferência Adotiva; Adulto; Animais; Encefalomielite Autoimune Experimental/genética; Feminino; Expressão Gênica; Humanos; Imunossupressores/uso terapêutico; Interferon Tipo I/metabolismo; Interferon gama/metabolismo; Interleucina-6/metabolismo; Camundongos; Camundongos Endogâmicos C57BL; Camundongos Knockout; Células Th1/imunologia; Células Th17/imunologia; Fator de Necrose Tumoral alfa/metabolismo; Proteínas tau/imunologia; Proteínas tau/uso terapêutico

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Fragmentos de Peptídeos / Encefalomielite Autoimune Experimental / Amiloide Limite: Adult / Animals / Female / Humans Idioma: En Revista: J Exp Med Ano de publicação: 2014 Tipo de documento: Article

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