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ILT3.Fc-CD166 Interaction Induces Inactivation of p70 S6 Kinase and Inhibits Tumor Cell Growth.
Xu, Zheng; Chang, Chih-Chao; Li, Muyang; Zhang, Qing-Yin; Vasilescu, Elena-Rodica M; D'Agati, Vivette; Floratos, Aristidis; Vlad, George; Suciu-Foca, Nicole.
Afiliação
  • Xu Z; Department of Pathology and Cell Biology, Columbia University, New York, NY 10032; and zx2142@columbia.edu.
  • Chang CC; Department of Pathology and Cell Biology, Columbia University, New York, NY 10032; and.
  • Li M; Department of Pathology and Cell Biology, Columbia University, New York, NY 10032; and.
  • Zhang QY; Department of Pathology and Cell Biology, Columbia University, New York, NY 10032; and.
  • Vasilescu EM; Department of Pathology and Cell Biology, Columbia University, New York, NY 10032; and.
  • D'Agati V; Department of Pathology and Cell Biology, Columbia University, New York, NY 10032; and.
  • Floratos A; Department of Systems Biology, Columbia University, New York, NY 10032.
  • Vlad G; Department of Pathology and Cell Biology, Columbia University, New York, NY 10032; and.
  • Suciu-Foca N; Department of Pathology and Cell Biology, Columbia University, New York, NY 10032; and.
J Immunol ; 200(3): 1207-1219, 2018 02 01.
Article em En | MEDLINE | ID: mdl-29263213

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Fragmentos Fc das Imunoglobulinas / Receptores de Superfície Celular / Molécula de Adesão de Leucócito Ativado / Proteínas Quinases S6 Ribossômicas 70-kDa / Proliferação de Células / Neoplasias Limite: Animals / Female / Humans Idioma: En Revista: J Immunol Ano de publicação: 2018 Tipo de documento: Article

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Fragmentos Fc das Imunoglobulinas / Receptores de Superfície Celular / Molécula de Adesão de Leucócito Ativado / Proteínas Quinases S6 Ribossômicas 70-kDa / Proliferação de Células / Neoplasias Limite: Animals / Female / Humans Idioma: En Revista: J Immunol Ano de publicação: 2018 Tipo de documento: Article
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