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Nuclear Receptor CAR Suppresses GADD45B-p38 MAPK Signaling to Promote Phenobarbital-induced Proliferation in Mouse Liver.
Hori, Takeshi; Saito, Kosuke; Moore, Rick; Flake, Gordon P; Negishi, Masahiko.
Afiliação
  • Hori T; Pharmacogenetics Section, Reproductive and Developmental Biology Laboratory, National Institute of Environmental Health Sciences, NIH, Research Triangle Park, North Carolina.
  • Saito K; Pharmacogenetics Section, Reproductive and Developmental Biology Laboratory, National Institute of Environmental Health Sciences, NIH, Research Triangle Park, North Carolina.
  • Moore R; Pharmacogenetics Section, Reproductive and Developmental Biology Laboratory, National Institute of Environmental Health Sciences, NIH, Research Triangle Park, North Carolina.
  • Flake GP; Cellular and Molecular Pathology Branch, Division of the National Toxicology Program, National Institute of Environmental Health Sciences, NIH, Research Triangle Park, North Carolina.
  • Negishi M; Pharmacogenetics Section, Reproductive and Developmental Biology Laboratory, National Institute of Environmental Health Sciences, NIH, Research Triangle Park, North Carolina. negishi@niehs.nih.gov.
Mol Cancer Res ; 16(8): 1309-1318, 2018 08.
Article em En | MEDLINE | ID: mdl-29716964
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Fenobarbital / Antígenos de Diferenciação / Receptores Citoplasmáticos e Nucleares / Proteínas Quinases p38 Ativadas por Mitógeno / Proliferação de Células / Fígado / Anticonvulsivantes Limite: Animals Idioma: En Revista: Mol Cancer Res Assunto da revista: BIOLOGIA MOLECULAR / NEOPLASIAS Ano de publicação: 2018 Tipo de documento: Article
Texto completo
Adicionar na Minha BVS
Imprimir
XML
PubMed Links
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Fenobarbital / Antígenos de Diferenciação / Receptores Citoplasmáticos e Nucleares / Proteínas Quinases p38 Ativadas por Mitógeno / Proliferação de Células / Fígado / Anticonvulsivantes Limite: Animals Idioma: En Revista: Mol Cancer Res Assunto da revista: BIOLOGIA MOLECULAR / NEOPLASIAS Ano de publicação: 2018 Tipo de documento: Article