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Self-reactive CD4+ IL-3+ T cells amplify autoimmune inflammation in myocarditis by inciting monocyte chemotaxis.
Anzai, Atsushi; Mindur, John E; Halle, Lennard; Sano, Soichi; Choi, Jennifer L; He, Shun; McAlpine, Cameron S; Chan, Christopher T; Kahles, Florian; Valet, Colin; Fenn, Ashley M; Nairz, Manfred; Rattik, Sara; Iwamoto, Yoshiko; Fairweather, DeLisa; Walsh, Kenneth; Libby, Peter; Nahrendorf, Matthias; Swirski, Filip K.
Afiliação
  • Anzai A; Center for Systems Biology, Massachusetts General Hospital and Harvard Medical School, Boston, MA.
  • Mindur JE; Department of Cardiology, Keio University School of Medicine, Tokyo, Japan.
  • Halle L; Center for Systems Biology, Massachusetts General Hospital and Harvard Medical School, Boston, MA.
  • Sano S; Center for Systems Biology, Massachusetts General Hospital and Harvard Medical School, Boston, MA.
  • Choi JL; Hematovascular Biology Center, Robert M. Berne Cardiovascular Research Center, University of Virginia School of Medicine, Charlottesville, VA.
  • He S; Center for Systems Biology, Massachusetts General Hospital and Harvard Medical School, Boston, MA.
  • McAlpine CS; Center for Systems Biology, Massachusetts General Hospital and Harvard Medical School, Boston, MA.
  • Chan CT; Center for Systems Biology, Massachusetts General Hospital and Harvard Medical School, Boston, MA.
  • Kahles F; Center for Systems Biology, Massachusetts General Hospital and Harvard Medical School, Boston, MA.
  • Valet C; Center for Systems Biology, Massachusetts General Hospital and Harvard Medical School, Boston, MA.
  • Fenn AM; Center for Systems Biology, Massachusetts General Hospital and Harvard Medical School, Boston, MA.
  • Nairz M; Center for Systems Biology, Massachusetts General Hospital and Harvard Medical School, Boston, MA.
  • Rattik S; Center for Systems Biology, Massachusetts General Hospital and Harvard Medical School, Boston, MA.
  • Iwamoto Y; Center for Systems Biology, Massachusetts General Hospital and Harvard Medical School, Boston, MA.
  • Fairweather D; Center for Systems Biology, Massachusetts General Hospital and Harvard Medical School, Boston, MA.
  • Walsh K; Department of Cardiovascular Medicine, Mayo Clinic, Jacksonville, FL.
  • Libby P; Hematovascular Biology Center, Robert M. Berne Cardiovascular Research Center, University of Virginia School of Medicine, Charlottesville, VA.
  • Nahrendorf M; Cardiovascular Division, Department of Medicine, Brigham and Women's Hospital, Boston, MA.
  • Swirski FK; Center for Systems Biology, Massachusetts General Hospital and Harvard Medical School, Boston, MA.
J Exp Med ; 216(2): 369-383, 2019 02 04.
Article em En | MEDLINE | ID: mdl-30670465
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Doenças Autoimunes / Monócitos / Linfócitos T CD4-Positivos / Quimiotaxia / Interleucina-3 / Miocardite Tipo de estudo: Prognostic_studies Limite: Animals Idioma: En Revista: J Exp Med Ano de publicação: 2019 Tipo de documento: Article País de afiliação: Marrocos
Texto completo
Adicionar na Minha BVS
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Doenças Autoimunes / Monócitos / Linfócitos T CD4-Positivos / Quimiotaxia / Interleucina-3 / Miocardite Tipo de estudo: Prognostic_studies Limite: Animals Idioma: En Revista: J Exp Med Ano de publicação: 2019 Tipo de documento: Article País de afiliação: Marrocos