Overexpression of NOX2 Exacerbates AngII-Mediated Cardiac Dysfunction and Metabolic Remodelling.

Hansen, Synne S; Pedersen, Tina M; Marin, Julie; Boardman, Neoma T; Shah, Ajay M; Aasum, Ellen; Hafstad, Anne D

Hansen, Synne S; Pedersen, Tina M; Marin, Julie; Boardman, Neoma T; Shah, Ajay M; Aasum, Ellen; Hafstad, Anne D.

Afiliação

Hansen SS; Cardiovascular Research Group, Department of Medical Biology, Faculty of Health Science, UiT-The Arctic University of Norway, 9019 Tromsø, Norway.
Pedersen TM; Cardiovascular Research Group, Department of Medical Biology, Faculty of Health Science, UiT-The Arctic University of Norway, 9019 Tromsø, Norway.
Marin J; Cardiovascular Research Group, Department of Medical Biology, Faculty of Health Science, UiT-The Arctic University of Norway, 9019 Tromsø, Norway.
Boardman NT; Cardiovascular Research Group, Department of Medical Biology, Faculty of Health Science, UiT-The Arctic University of Norway, 9019 Tromsø, Norway.
Shah AM; School of Cardiovascular Medicine & Sciences, King's College London, British Heart Foundation Centre of Excellence, London SE5 9NU, UK.
Aasum E; Cardiovascular Research Group, Department of Medical Biology, Faculty of Health Science, UiT-The Arctic University of Norway, 9019 Tromsø, Norway.
Hafstad AD; Cardiovascular Research Group, Department of Medical Biology, Faculty of Health Science, UiT-The Arctic University of Norway, 9019 Tromsø, Norway.

Antioxidants (Basel) ; 11(1)2022 Jan 10.

Article em En | MEDLINE | ID: mdl-35052647

Palavras-chave

NOX2; angiotensin II; cardiac disease; cardiac efficiency; cardiac hypertrophy; hypertension

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Idioma: En Revista: Antioxidants (Basel) Ano de publicação: 2022 Tipo de documento: Article País de afiliação: Noruega

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