CX3CL1 deficiency ameliorates inflammation, apoptosis and accelerates osteogenic differentiation, mineralization in LPS-treated MC3T3-E1 cells via its receptor CX3CR1.

Lu, Zhihua; Zhang, Aihua; Dai, Yan

Lu, Zhihua; Zhang, Aihua; Dai, Yan.

Afiliação

Lu Z; Medical school, Yangzhou Polytechnic College, Yangzhou, Jiangsu 225009, China.
Zhang A; Department of Rehabilitation, Northern Jiangsu People's Hospital, Yangzhou, Jiangsu 225001, China; Clinical Medical College of Yangzhou University, Yangzhou, Jiangsu 225009, China.
Dai Y; Medical research center, Northern Jiangsu People's Hospital, Yangzhou, Jiangsu 225001, China; Clinical Medical College of Yangzhou University, Yangzhou, Jiangsu 225009, China. Electronic address: daidyan@126.com.

Ann Anat ; 246: 152036, 2023 Feb.

Article em En | MEDLINE | ID: mdl-36436718

Assuntos

Calcinose; Quimiocinas CX3C; Humanos; Osteogênese; Lipopolissacarídeos/toxicidade; Ligantes; Linhagem Celular; Diferenciação Celular; Osteoblastos; Apoptose; Quimiocinas CX3C/metabolismo; Receptor 1 de Quimiocina CX3C/metabolismo; Quimiocina CX3CL1/metabolismo

Palavras-chave

CX3CL1; CX3CR1; Inflammation; Mineralization; Osteoblast differentiation; Osteoporosis

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Contexto em Saúde: 4_TD / 6_ODS3_enfermedades_notrasmisibles Problema de saúde: 4_aids / 6_endocrine_disorders / 6_musculoskeletal_diseases_rheumatic_disorders Assunto principal: Calcinose / Quimiocinas CX3C Limite: Humans Idioma: En Revista: Ann Anat Assunto da revista: ANATOMIA Ano de publicação: 2023 Tipo de documento: Article País de afiliação: China

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