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Oncotarget ; 7(40): 65042-65051, 2016 Oct 04.
Artigo em Inglês | MEDLINE | ID: mdl-27542255

RESUMO

OBJECTIVE: Gene therapy is a frontier in modern medicine. In the present study, we explored a new technique for the effective treatment of multidrug-resistant (MDR) breast cancer by combining fully the advantages of multidisciplinary fields, including image-guided minimally invasive interventional oncology, radiofrequency technology, and direct intratumoral gene therapy. RESULTS: Combination treatment with PHSP-TK plus RFH resulted in significantly higher TK gene transfection/expression, as well as a lower cell proliferation rate and a higher cell apoptosis index, than those of control groups. In vivo validation experiments with MRI confirmed that combination therapy resulted in a significant reduction of relative tumor volume compared with those of control animals, which was supported by the results of histologic and apoptosis analyses. MATERIALS AND METHODS: The heat shock protein promoter (PHSP) was used to precisely control the overexpression of thymidine kinase (TK) (PHSP-TK). Serial in vitro experiments were performed to confirm whether radiofrequency hyperthermia (RFH) could enhance PHSP-TK transfection and expression in a MDR breast cancer cell line (MCF7/Adr). Serial in vivo experiments were then carried out to validate the feasibility of the new technique, termed interventional RFH-enhanced direct intratumoral PHSP-TK gene therapy. The therapeutic effect of combination therapy was evaluated by MRI and confirmed by subsequent laboratory correlation. CONCLUSIONS: This study has established "proof-of-principle" of a new technique, interventional RFH-enhanced local gene therapy for MDR breast cancer, which may open new avenues for the effective management of MDR breast cancers via the simultaneous integration of interventional oncology, RF technology, and direct intratumoral gene therapy.


Assuntos
Neoplasias da Mama , Terapia Genética/métodos , Hipertermia Induzida/métodos , Timidina Quinase/genética , Transfecção/métodos , Animais , Resistência a Múltiplos Medicamentos , Resistencia a Medicamentos Antineoplásicos , Feminino , Genes Transgênicos Suicidas , Proteínas de Choque Térmico/genética , Humanos , Células MCF-7 , Camundongos , Camundongos Endogâmicos BALB C , Regiões Promotoras Genéticas , Timidina Quinase/administração & dosagem , Timidina Quinase/biossíntese , Ensaios Antitumorais Modelo de Xenoenxerto
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