1.
Eur J Immunol
; 51(2): 490-493, 2021 02.
Artigo
em Inglês
| MEDLINE
| ID: mdl-33022775
RESUMO
We show that the intraerythrocytic stages of the malaria parasite Plasmodium falciparum bind plasminogen and mediate its conversion into plasmin to inactivate parasite-bound C3b. This complement evasion mechanism counteracts terminal complex formation and hence promotes parasite survival in human blood.