RESUMO
The present study explored the types of errors found in Google Translate (GT) Chinese-to-English translations and, based on those error types, proposes strategies for optimizing the performance of GT. Seven abstracts written in both Chinese and English from seven articles published in English Teaching and Learning in 2017 were selected as the materials. The researchers compared the GT translations to the English abstracts written by the original author(s) and analyzed the problems in the translations. The problematic translations consisted of grammatical errors (35%) and lexical errors (65%). Relatedly, we propose nine specific strategies to employ when writing Chinese abstracts to be translated into English using GT. According to the strategies, we suggest that users (1) avoid native language-specific expressions, (2) maintain the use of original English terminologies in composing Chinese abstracts, and (3) enhance logical relations and expressions for the discipline-specific discourse community. Further analyses revealed that 99% of the 69 identified problems in the GT translations of the seven abstracts could be avoided by using the proposed strategies. A conceptual framework for the collaboration between GT and GT users is proposed and pedagogical implications are discussed.
RESUMO
Insect growth and development are precisely controlled by hormone homeostasis. The prothoracicotropic hormone (PTTH) receptor, Torso, is a member of the receptor tyrosine kinase family in insects. Activation of Torso by PTTH triggers biosynthesis and release of the steroid hormone in the prothoracic gland (PG). Although numbers of genes functioning in steroid hormone synthesis and metabolism have been identified in insects, the PTTH transduction pathway via its receptor Torso is poorly understood. In the current study, we describe a loss-of-function analysis of Torso in the silkworm, Bombyx mori, by targeted gene disruption using the transgenic CRISPR/Cas9 (clustered regularly interspaced short palindromic repeats/RNA-guided Cas9 nucleases) system. Depletion of B. mori Torso (BmTorso) did not eventually affect larval ecdysis and metamorphosis processes. Instead, BmTorso deficiency resulted in significant extension of developing time during larval and pupal stages with increased pupa and cocoon sizes. The ecdysteriod titers in the hemolymph of BmTorso mutants sharpy declined. Transcriptional levels of genes involved in ecdysone biosynthesis and ecdysteroid signaling pathways were significantly reduced in BmTorso-deficient animals. Additionally, RNA-Seq analysis revealed that genes involved in the longevity pathway and protein processing in the endoplasmic reticulum pathway were affected after BmTorso deletion. These results indicate that Torso is critical for maintaining steroid hormone homeostasis in insects.