Hypothermia during reperfusion after asphyxial cardiac arrest improves functional recovery and selectively alters stress-induced protein expression.

Hicks, S D; DeFranco, D B; Callaway, C W

Hicks, S D; DeFranco, D B; Callaway, C W.

Affiliation

Hicks SD; Department of Emergency Medicine, University of Pittsburgh, Pennsylvania, USA.

J Cereb Blood Flow Metab ; 20(3): 520-30, 2000 Mar.

Article in En | MEDLINE | ID: mdl-10724117

Subject(s)

Asphyxia/complications; HSP70 Heat-Shock Proteins/metabolism; Heart Arrest/etiology; Hypothermia, Induced; Myocardial Reperfusion Injury/therapy; Animals; Cerebellum/metabolism; Cerebellum/pathology; Hippocampus/metabolism; Hippocampus/pathology; Male; Motor Activity; Myocardial Reperfusion Injury/mortality; Myocardial Reperfusion Injury/physiopathology; Neurons/metabolism; Rats; Rats, Sprague-Dawley; Time Factors

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Collection: 01-internacional Database: MEDLINE Main subject: Asphyxia / Myocardial Reperfusion Injury / HSP70 Heat-Shock Proteins / Heart Arrest / Hypothermia, Induced Limits: Animals Language: En Journal: J Cereb Blood Flow Metab Year: 2000 Document type: Article

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