Hypothermia differentially increases extracellular signal-regulated kinase and stress-activated protein kinase/c-Jun terminal kinase activation in the hippocampus during reperfusion after asphyxial cardiac arrest.

Hicks, S D; Parmele, K T; DeFranco, D B; Klann, E; Callaway, C W

Hicks, S D; Parmele, K T; DeFranco, D B; Klann, E; Callaway, C W.

Affiliation

Hicks SD; Department of Emergency Medicine, University of Pittsburgh, 230 McKee Place, Suite 400, Pittsburgh, PA 15213, USA.

Neuroscience ; 98(4): 677-85, 2000.

Article in En | MEDLINE | ID: mdl-10891611

Subject(s)

Asphyxia/metabolism; Heart Arrest/metabolism; Hypothermia/metabolism; Mitogen-Activated Protein Kinase Kinases/metabolism; Proto-Oncogene Proteins c-jun/metabolism; Animals; Enzyme Induction; Hippocampus/metabolism; Phosphorylation; Rats; Reperfusion

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Collection: 01-internacional Health context: 6_ODS3_enfermedades_notrasmisibles Database: MEDLINE Main subject: Asphyxia / Proto-Oncogene Proteins c-jun / Mitogen-Activated Protein Kinase Kinases / Heart Arrest / Hypothermia Limits: Animals Language: En Journal: Neuroscience Year: 2000 Document type: Article

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