Fas receptor and neuronal cell death after spinal cord ischemia.

Matsushita, K; Wu, Y; Qiu, J; Lang-Lazdunski, L; Hirt, L; Waeber, C; Hyman, B T; Yuan, J; Moskowitz, M A

Matsushita, K; Wu, Y; Qiu, J; Lang-Lazdunski, L; Hirt, L; Waeber, C; Hyman, B T; Yuan, J; Moskowitz, M A.

Affiliation

Matsushita K; Neuroscience Center, Massachusetts General Hospital, Harvard Medical School, Boston, Massachusetts 02129, USA.

J Neurosci ; 20(18): 6879-87, 2000 Sep 15.

Article in En | MEDLINE | ID: mdl-10995832

Subject(s)

Apoptosis; Neurons/metabolism; Spinal Cord Ischemia/metabolism; Spinal Cord/metabolism; fas Receptor/metabolism; Animals; Caspase 3; Caspase 8; Caspase 9; Caspases/genetics; Caspases/metabolism; Cytochrome c Group/metabolism; Disease Models, Animal; Enzyme Precursors/genetics; Enzyme Precursors/metabolism; Female; Gelsolin/metabolism; In Situ Nick-End Labeling; Male; Mice; Mice, Inbred C57BL; Neurons/pathology; RNA, Messenger/biosynthesis; Reperfusion; Signal Transduction; Spinal Cord/blood supply; Spinal Cord/pathology; Spinal Cord Ischemia/pathology

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Full text: 1 Collection: 01-internacional Health context: 6_ODS3_enfermedades_notrasmisibles Database: MEDLINE Main subject: Spinal Cord / Apoptosis / Fas Receptor / Spinal Cord Ischemia / Neurons Type of study: Prognostic_studies Limits: Animals Language: En Journal: J Neurosci Year: 2000 Document type: Article

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