Mechanisms underlying the inability to induce area CA1 LTP in the mouse after traumatic brain injury.

Schwarzbach, E; Bonislawski, D P; Xiong, G; Cohen, A S

Schwarzbach, E; Bonislawski, D P; Xiong, G; Cohen, A S.

Affiliation

Schwarzbach E; Division of Neurology, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania 19104, USA. eschwarz@mail.med.upenn.edu

Hippocampus ; 16(6): 541-50, 2006.

Article in En | MEDLINE | ID: mdl-16634077

Subject(s)

Brain Injuries/complications; Brain Injuries/physiopathology; Hippocampus/physiopathology; Long-Term Potentiation; Memory Disorders/etiology; Memory Disorders/physiopathology; Animals; Brain Injuries/metabolism; Calcium-Calmodulin-Dependent Protein Kinase Type 2; Calcium-Calmodulin-Dependent Protein Kinases/metabolism; Dendritic Spines/drug effects; Dendritic Spines/metabolism; Dendritic Spines/ultrastructure; Disease Models, Animal; Excitatory Postsynaptic Potentials/drug effects; Glutamic Acid/metabolism; Glutamic Acid/pharmacology; Hippocampus/metabolism; Long-Term Synaptic Depression; Memory Disorders/metabolism; Mice; Mice, Inbred C57BL; Neural Pathways/metabolism; Neural Pathways/physiopathology; Organ Culture Techniques; Receptors, N-Methyl-D-Aspartate/drug effects; Receptors, N-Methyl-D-Aspartate/metabolism; Synaptic Transmission

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Brain Injuries / Long-Term Potentiation / Hippocampus / Memory Disorders Type of study: Prognostic_studies Limits: Animals Language: En Journal: Hippocampus Year: 2006 Document type: Article

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