Different mechanisms account for extracellular-signal regulated kinase activation in distinct brain regions following global ischemia and reperfusion.

Ho, Y; Logue, E; Callaway, C W; DeFranco, D B

Ho, Y; Logue, E; Callaway, C W; DeFranco, D B.

Affiliation

Ho Y; Center for Neuroscience, University of Pittsburgh School of Medicine, 200 Lothrop Street, Pittsburgh, PA 15261, USA.

Neuroscience ; 145(1): 248-55, 2007 Mar 02.

Article in En | MEDLINE | ID: mdl-17207579

Subject(s)

Brain/enzymology; Extracellular Signal-Regulated MAP Kinases/metabolism; Ischemia/enzymology; Ischemia/pathology; Reperfusion; Animals; Blotting, Western/methods; Brain/drug effects; Brain/pathology; Disease Models, Animal; Enzyme Activation/drug effects; Enzyme Inhibitors/pharmacology; Male; Okadaic Acid/pharmacology; Phosphoric Monoester Hydrolases/metabolism; Rats; Rats, Sprague-Dawley

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Brain / Reperfusion / Extracellular Signal-Regulated MAP Kinases / Ischemia Limits: Animals Language: En Journal: Neuroscience Year: 2007 Document type: Article

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