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Biliverdin modulates the expression of C5aR in response to endotoxin in part via mTOR signaling.
Bisht, Kavita; Wegiel, Barbara; Tampe, Jens; Neubauer, Oliver; Wagner, Karl-Heinz; Otterbein, Leo E; Bulmer, Andrew C.
Affiliation
  • Bisht K; Heart Foundation Research Center, Griffith Health Institute, Griffith University, Gold Coast, Australia; Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, USA.
  • Wegiel B; Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, USA.
  • Tampe J; Griffith Enterprise, Griffith University, Nathan, Australia.
  • Neubauer O; Emerging Field Oxidative Stress and DNA Stability and Research Platform Active Aging, Department of Nutritional Science, University of Vienna, Vienna, Austria.
  • Wagner KH; Emerging Field Oxidative Stress and DNA Stability and Research Platform Active Aging, Department of Nutritional Science, University of Vienna, Vienna, Austria.
  • Otterbein LE; Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, USA.
  • Bulmer AC; Heart Foundation Research Center, Griffith Health Institute, Griffith University, Gold Coast, Australia. Electronic address: a.bulmer@griffith.edu.au.
Biochem Biophys Res Commun ; 449(1): 94-9, 2014 Jun 20.
Article in En | MEDLINE | ID: mdl-24814708
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Biliverdine / Lipopolysaccharides / Receptor, Anaphylatoxin C5a / TOR Serine-Threonine Kinases / Macrophage Activation / Macrophages Limits: Animals Language: En Journal: Biochem Biophys Res Commun Year: 2014 Document type: Article
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Biliverdine / Lipopolysaccharides / Receptor, Anaphylatoxin C5a / TOR Serine-Threonine Kinases / Macrophage Activation / Macrophages Limits: Animals Language: En Journal: Biochem Biophys Res Commun Year: 2014 Document type: Article