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Neuronal Apolipoprotein E4 Expression Results in Proteome-Wide Alterations and Compromises Bioenergetic Capacity by Disrupting Mitochondrial Function.
Orr, Adam L; Kim, Chaeyoung; Jimenez-Morales, David; Newton, Billy W; Johnson, Jeffrey R; Krogan, Nevan J; Swaney, Danielle L; Mahley, Robert W.
Affiliation
  • Orr AL; Gladstone Institute of Neurological Disease, San Francisco, CA, USA.
  • Kim C; Present address: Helen & Robert Appel Alzheimer's Disease Research Institute, Feil Family Brain and Mind Research Institute, Weill Cornell Medicine, New York, NY, USA.
  • Jimenez-Morales D; Gladstone Institute of Neurological Disease, San Francisco, CA, USA.
  • Newton BW; Gladstone Institute of Data Science and Biotechnology, San Francisco, CA, USA.
  • Johnson JR; Department of Cellular and Molecular Pharmacology, University of California, San Francisco, CA, USA.
  • Krogan NJ; Present address: Department of Medicine, Division of Cardiovascular Medicine, Stanford University, CA, USA.
  • Swaney DL; Department of Cellular and Molecular Pharmacology, University of California, San Francisco, CA, USA.
  • Mahley RW; Gladstone Institute of Data Science and Biotechnology, San Francisco, CA, USA.
J Alzheimers Dis ; 68(3): 991-1011, 2019.
Article in En | MEDLINE | ID: mdl-30883359
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Proteome / Apolipoprotein E4 / Mitochondria / Neurons Type of study: Risk_factors_studies Limits: Animals Language: En Journal: J Alzheimers Dis Year: 2019 Document type: Article
Fulltext
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PubMed Links
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Proteome / Apolipoprotein E4 / Mitochondria / Neurons Type of study: Risk_factors_studies Limits: Animals Language: En Journal: J Alzheimers Dis Year: 2019 Document type: Article