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Genetic compensation in a stable slc25a46 mutant zebrafish: A case for using F0 CRISPR mutagenesis to study phenotypes caused by inherited disease.
Buglo, Elena; Sarmiento, Evan; Martuscelli, Nicole Belliard; Sant, David W; Danzi, Matt C; Abrams, Alexander J; Dallman, Julia E; Züchner, Stephan.
Affiliation
  • Buglo E; John P. Hussman Institute for Human Genomics, University of Miami Miller School of Medicine, Miami, Florida, United States of America.
  • Sarmiento E; Department of Biological Sciences, University of Miami, Coral Gables, Florida, United States of America.
  • Martuscelli NB; Department of Biological Sciences, University of Miami, Coral Gables, Florida, United States of America.
  • Sant DW; Department of Biomedical Informatics, University of Utah, Salt Lake City, Utah, United States of America.
  • Danzi MC; John P. Hussman Institute for Human Genomics, University of Miami Miller School of Medicine, Miami, Florida, United States of America.
  • Abrams AJ; Department of Genetics, Yale University, New Haven, Connecticut, United States of America.
  • Dallman JE; Department of Biological Sciences, University of Miami, Coral Gables, Florida, United States of America.
  • Züchner S; John P. Hussman Institute for Human Genomics, University of Miami Miller School of Medicine, Miami, Florida, United States of America.
PLoS One ; 15(3): e0230566, 2020.
Article in En | MEDLINE | ID: mdl-32208444
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Zebrafish / Zebrafish Proteins / Mitochondrial Membrane Transport Proteins / CRISPR-Cas Systems Limits: Animals Language: En Journal: PLoS One Year: 2020 Document type: Article
Fulltext
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PubMed Links
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Zebrafish / Zebrafish Proteins / Mitochondrial Membrane Transport Proteins / CRISPR-Cas Systems Limits: Animals Language: En Journal: PLoS One Year: 2020 Document type: Article