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Intestinal Inflammation Promotes MDL-1+ Osteoclast Precursor Expansion to Trigger Osteoclastogenesis and Bone Loss.
Peek, Christopher T; Ford, Caleb A; Eichelberger, Kara R; Jacobse, Justin; Torres, Teresa P; Maseda, Damian; Latour, Yvonne L; Piazuelo, M Blanca; Johnson, Joshua R; Byndloss, Mariana X; Wilson, Keith T; Rathmell, Jeffrey C; Goettel, Jeremy A; Cassat, James E.
Affiliation
  • Peek CT; Department of Pathology, Microbiology, and Immunology, Vanderbilt University Medical Center, Nashville, Tennessee.
  • Ford CA; Department of Biomedical Engineering, Vanderbilt University, Nashville, Tennessee.
  • Eichelberger KR; Department of Pediatrics, Division of Pediatric Infectious Diseases, Vanderbilt University Medical Center, Nashville, Tennessee.
  • Jacobse J; Department of Pathology, Microbiology, and Immunology, Vanderbilt University Medical Center, Nashville, Tennessee; Division of Gastroenterology, Hepatology, and Nutrition, Department of Medicine, Vanderbilt University Medical Center, Nashville, Tennessee.
  • Torres TP; Department of Pathology, Microbiology, and Immunology, Vanderbilt University Medical Center, Nashville, Tennessee.
  • Maseda D; Department of Pathology, Microbiology, and Immunology, Vanderbilt University Medical Center, Nashville, Tennessee; Department of Dermatology, University of Pennsylvania, Philadelphia, Pennsylvania.
  • Latour YL; Department of Pathology, Microbiology, and Immunology, Vanderbilt University Medical Center, Nashville, Tennessee.
  • Piazuelo MB; Division of Gastroenterology, Hepatology, and Nutrition, Department of Medicine, Vanderbilt University Medical Center, Nashville, Tennessee; Center for Mucosal Inflammation and Cancer, Vanderbilt University Medical Center, Nashville, Tennessee.
  • Johnson JR; Vanderbilt Center for Bone Biology, Vanderbilt University Medical Center, Nashville, Tennessee.
  • Byndloss MX; Department of Pathology, Microbiology, and Immunology, Vanderbilt University Medical Center, Nashville, Tennessee; Vanderbilt Institute for Infection, Immunology, and Inflammation, Vanderbilt University Medical Center, Nashville, Tennessee.
  • Wilson KT; Department of Pathology, Microbiology, and Immunology, Vanderbilt University Medical Center, Nashville, Tennessee; Division of Gastroenterology, Hepatology, and Nutrition, Department of Medicine, Vanderbilt University Medical Center, Nashville, Tennessee; Vanderbilt Institute for Infection, Immunolo
  • Rathmell JC; Department of Pathology, Microbiology, and Immunology, Vanderbilt University Medical Center, Nashville, Tennessee; Vanderbilt Institute for Infection, Immunology, and Inflammation, Vanderbilt University Medical Center, Nashville, Tennessee; Center for Mucosal Inflammation and Cancer, Vanderbilt Univ
  • Goettel JA; Department of Pathology, Microbiology, and Immunology, Vanderbilt University Medical Center, Nashville, Tennessee; Division of Gastroenterology, Hepatology, and Nutrition, Department of Medicine, Vanderbilt University Medical Center, Nashville, Tennessee; Vanderbilt Institute for Infection, Immunolo
  • Cassat JE; Department of Pathology, Microbiology, and Immunology, Vanderbilt University Medical Center, Nashville, Tennessee; Department of Biomedical Engineering, Vanderbilt University, Nashville, Tennessee; Department of Pediatrics, Division of Pediatric Infectious Diseases, Vanderbilt University Medical Cen
Cell Mol Gastroenterol Hepatol ; 14(4): 731-750, 2022.
Article in En | MEDLINE | ID: mdl-35835390
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Osteoclasts / Osteogenesis / Bone Resorption / Inflammatory Bowel Diseases / Receptors, Cell Surface / Lectins, C-Type Limits: Animals Language: En Journal: Cell Mol Gastroenterol Hepatol Year: 2022 Document type: Article
Fulltext
Print
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PubMed Links
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Osteoclasts / Osteogenesis / Bone Resorption / Inflammatory Bowel Diseases / Receptors, Cell Surface / Lectins, C-Type Limits: Animals Language: En Journal: Cell Mol Gastroenterol Hepatol Year: 2022 Document type: Article