Intracellular IL-1 alpha in fibroblasts as a possible endogenous mediator of joint damage in rheumatoid arthritis.

Apte, R N

Apte, R N.

Affiliation

Apte RN; Department of Microbiology and Immunology, Faculty of Health Sciences, Ben-Gurion University of the Negev, Beer Sheva, Israel.

Isr J Med Sci ; 33(4): 285-8, 1997 Apr.

Article in En | MEDLINE | ID: mdl-9347881

Subject(s)

Arthritis, Rheumatoid/immunology; Autoimmune Diseases/immunology; Fibroblasts/immunology; Inflammation Mediators/immunology; Interleukin-1/immunology; Joints/immunology; Animals; Cell Division/immunology; Cellular Senescence/immunology; Gene Expression Regulation; Humans; Interleukin 1 Receptor Antagonist Protein; Interleukin-1/genetics; Receptors, Interleukin-1/antagonists & inhibitors; Receptors, Interleukin-1/immunology; Sialoglycoproteins/immunology

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Collection: 01-internacional Database: MEDLINE Main subject: Arthritis, Rheumatoid / Autoimmune Diseases / Interleukin-1 / Inflammation Mediators / Fibroblasts / Joints Limits: Animals / Humans Language: En Journal: Isr J Med Sci Year: 1997 Document type: Article

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